Kaisa Silander

32.1k citations
70 papers · 3.1k · h-index 35

Impact in

Papers in

    • Genetic Associations and Epidemiology 21
    • Genomic variations and chromosomal abnormalities 8
    • BRCA gene mutations in cancer 4

Kaisa Silander

69 papers receiving 3.0k citations

Peers

Kaisa Silander
Comparison fields: 5 of 130
  • Genetics 827
  • Endocrine and Autonomic Systems 161
  • Biological Psychiatry 46
  • Cellular and Molecular Neuroscience 323
  • Endocrinology, Diabetes and Metabolism 246
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Citations per field
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Citations per year

Countries citing papers authored by Kaisa Silander

Since Specialization
Citations

This map shows the geographic impact of Kaisa Silander's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kaisa Silander with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kaisa Silander more than expected).

Fields of papers citing papers by Kaisa Silander

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kaisa Silander. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kaisa Silander. The network helps show where Kaisa Silander may publish in the future.

Co-authors

The 25 scholars most cited alongside Kaisa Silander, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Kaisa Silander Line = papers co-authored together Kaisa Silander links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 70 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2010389
2 2010169
3 2012166
4 2003113
5 2008108
6 199998
7 200797
8 200288
9 200787
10 200783
11 201079
12 199777
13 200473
14 200369
15 199869
16 200867
17 201167
18 201066
19 200163
20 200662

About Kaisa Silander

Kaisa Silander is a scholar working on Genetics, Molecular Biology, Cellular and Molecular Neuroscience, Surgery and Epidemiology, having authored 70 papers that have together received 3.1k indexed citations. Recurring topics across this work include Genetic Associations and Epidemiology (21 papers), Hereditary Neurological Disorders (8 papers), Genomic variations and chromosomal abnormalities (8 papers), Genetic Neurodegenerative Diseases (6 papers), Adipokines, Inflammation, and Metabolic Diseases (4 papers), BRCA gene mutations in cancer (4 papers), Ethics in Clinical Research (4 papers) and Helicobacter pylori-related gastroenterology studies (3 papers). The work is most often cited by research in Genetics (827 citations), Endocrine and Autonomic Systems (161 citations), Biological Psychiatry (46 citations), Cellular and Molecular Neuroscience (323 citations) and Endocrinology, Diabetes and Metabolism (246 citations). Kaisa Silander has collaborated with scholars based in Finland, United States and United Kingdom. Frequent co-authors include Veikko Salomaa, Markus Perola, Leena Peltonen, Leena Peltonen, Samuli Ripatti, Timo Partonen, Janna Saarela, Jouko Lönnqvist, Pekka Jousilahti and Tiina Paunio. Their work appears in journals such as PLoS Genetics, Human Mutation, Human Molecular Genetics, Biological Psychiatry and Human Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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