Josefa Rius
Impact in
- Hematology top 10%
- Autoimmune and Inflammatory Disorders Research
- Immunology top 10%
- IL-33, ST2, and ILC Pathways
- interferon and immune responses
- Immune Cell Function and Interaction
Papers in
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- Inflammasome and immune disorders 6
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- IL-33, ST2, and ILC Pathways 3
- Co-authors
- Jordi Yagüe (8 shared papers)Juan I. Aróstegui (8 shared papers)Susana Plaza (6 shared papers)Anna Aldea (4 shared papers)Jordi Vives (4 shared papers)Consuelo Modesto (2 shared papers)Eduardo Ramos (2 shared papers)Rosa Merino (1 shared paper)
- Journals
- Human Mutation (1 paper)European Journal of Pediatrics (1 paper)American Journal of Medical Genetics Part A (1 paper)Arthritis & Rheumatism (4 papers)PubMed (1 paper)
- Partner nations
- SpainUnited States
In The Last Decade
Josefa Rius
8 papers receiving 514 citations
Peers
Comparison fields: 5 of 47
- Hematology 143
- Immunology 245
- Nephrology 75
- Rheumatology 93
- Molecular Biology 380
Countries citing papers authored by Josefa Rius
This map shows the geographic impact of Josefa Rius's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Josefa Rius with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Josefa Rius more than expected).
Fields of papers citing papers by Josefa Rius
This network shows the impact of papers produced by Josefa Rius. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Josefa Rius. The network helps show where Josefa Rius may publish in the future.
Co-authors
The 25 scholars most cited alongside Josefa Rius, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2007 | 165 | |
| 2 | 2004 | 73 | |
| 3 | 2003 | 65 | |
| 4 | 2010 | 55 | |
| 5 | Effect of NOD2/CARD15 variants in T-cell depleted allogeneic stem cell transplantation. | 2006 | 51 |
| 6 | 2007 | 51 | |
| 7 | 2004 | 35 | |
| 8 | 2004 | 33 |
About Josefa Rius
Josefa Rius is a scholar working on Molecular Biology, Immunology, Hematology, Nephrology and Rheumatology, having authored 8 papers that have together received 528 indexed citations. Recurring topics across this work include Inflammasome and immune disorders (6 papers), IL-33, ST2, and ILC Pathways (3 papers), Autoimmune and Inflammatory Disorders Research (2 papers), Eosinophilic Disorders and Syndromes (2 papers), Gout, Hyperuricemia, Uric Acid (2 papers), Sarcoidosis and Beryllium Toxicity Research (1 paper), Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (1 paper) and Hematopoietic Stem Cell Transplantation (1 paper). The work is most often cited by research in Hematology (143 citations), Immunology (245 citations), Nephrology (75 citations), Rheumatology (93 citations) and Molecular Biology (380 citations). Josefa Rius has collaborated with scholars based in Spain and United States. Frequent co-authors include Jordi Yagüe, Juan I. Aróstegui, Susana Plaza, Anna Aldea, Jordi Vives, Consuelo Modesto, Eduardo Ramos, Rosa Merino, Antonio Naranjo and Julia García-Consuegra. Their work appears in journals such as Human Mutation, European Journal of Pediatrics, American Journal of Medical Genetics Part A, Arthritis & Rheumatism and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.