John J. Acton

566 citations
18 papers · 409 · h-index 10

Impact in

  • Biochemistry top 10%
    • Eicosanoids and Hypertension Pharmacology
    • Peroxisome Proliferator-Activated Receptors
    • Metabolism, Diabetes, and Cancer
    • Receptor Mechanisms and Signaling

Papers in

    • Peroxisome Proliferator-Activated Receptors 4
    • Chemical Synthesis and Analysis 3
    • Cancer therapeutics and mechanisms 2
    • Plant biochemistry and biosynthesis 2

John J. Acton

17 papers receiving 380 citations

Peers

John J. Acton
Comparison fields: 5 of 64
  • Biochemistry 52
  • Molecular Biology 282
  • Physiology 106
  • Pharmacology 66
  • Cellular and Molecular Neuroscience 61
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Countries citing papers authored by John J. Acton

Since Specialization
Citations

This map shows the geographic impact of John J. Acton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John J. Acton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John J. Acton more than expected).

Fields of papers citing papers by John J. Acton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John J. Acton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John J. Acton. The network helps show where John J. Acton may publish in the future.

Co-authors

The 25 scholars most cited alongside John J. Acton, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John J. Acton Line = papers co-authored together John J. Acton links everyone, so they are left out of the graph.

All Works

18 of 18 papers shown
#Work
1 2004105
2 200556
3 198849
4 200947
5 199245
6 199222
7 201021
8 199419
9 199113
10 199612
11 19894
12 19993
13 19933
14 20133
15 19883
16 19993
17 19941
18 19960

About John J. Acton

John J. Acton is a scholar working on Molecular Biology, Organic Chemistry, Physiology, Cellular and Molecular Neuroscience and Pharmacology, having authored 18 papers that have together received 409 indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (4 papers), Neuropeptides and Animal Physiology (3 papers), Adipose Tissue and Metabolism (3 papers), Chemical Synthesis and Analysis (3 papers), Cancer therapeutics and mechanisms (2 papers), Plant biochemistry and biosynthesis (2 papers), Nitric Oxide and Endothelin Effects (2 papers) and Pharmacological Effects of Natural Compounds (2 papers). The work is most often cited by research in Biochemistry (52 citations), Molecular Biology (282 citations), Physiology (106 citations), Pharmacology (66 citations) and Cellular and Molecular Neuroscience (61 citations). John J. Acton has collaborated with scholars based in United States and Canada. Frequent co-authors include A. Brian Jones, Thomas W. Doebber, Harold B. Wood, Joel P. Berger, David E. Moller, Lawrence F. Colwell, Ronald Melzack, Karen L. MacNaul, Regina M. Black and John E. McKenna. Their work appears in journals such as Bioorganic & Medicinal Chemistry Letters, Journal of Medicinal Chemistry, Tetrahedron Letters, Experimental Neurology and Biochemical and Biophysical Research Communications.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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