John D. H. Stead

1.8k citations
27 papers · 1.3k · h-index 19

Impact in

Papers in

    • DNA Repair Mechanisms 2
    • Gene expression and cancer classification 2
    • Genetic Associations and Epidemiology 4
    • Genetic Mapping and Diversity in Plants and Animals 3

John D. H. Stead

22 papers receiving 1.3k citations

Peers

John D. H. Stead
Comparison fields: 5 of 99
  • Biological Psychiatry 171
  • Behavioral Neuroscience 222
  • Developmental Neuroscience 119
  • Cellular and Molecular Neuroscience 257
  • Social Psychology 259
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Citations per field
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Citations per year

Countries citing papers authored by John D. H. Stead

Since Specialization
Citations

This map shows the geographic impact of John D. H. Stead's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John D. H. Stead with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John D. H. Stead more than expected).

Fields of papers citing papers by John D. H. Stead

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John D. H. Stead. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John D. H. Stead. The network helps show where John D. H. Stead may publish in the future.

Co-authors

The 25 scholars most cited alongside John D. H. Stead, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John D. H. Stead Line = papers co-authored together John D. H. Stead links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 27 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2004316
2 2006163
3 2013126
4 2007109
5 200679
6 202253
7 199950
8 200849
9 200046
10 201145
11 200043
12 199733
13 200331
14 201031
15 201231
16 200228
17 199825
18 201323
19 201320
20 202013

About John D. H. Stead

John D. H. Stead is a scholar working on Molecular Biology, Genetics, Social Psychology, Surgery and Clinical Psychology, having authored 27 papers that have together received 1.3k indexed citations. Recurring topics across this work include Neuroendocrine regulation and behavior (5 papers), Genetic Associations and Epidemiology (4 papers), Neurogenesis and neuroplasticity mechanisms (3 papers), Genetic Mapping and Diversity in Plants and Animals (3 papers), Pancreatic function and diabetes (3 papers), Stress Responses and Cortisol (3 papers), DNA Repair Mechanisms (2 papers) and Gene expression and cancer classification (2 papers). The work is most often cited by research in Biological Psychiatry (171 citations), Behavioral Neuroscience (222 citations), Developmental Neuroscience (119 citations), Cellular and Molecular Neuroscience (257 citations) and Social Psychology (259 citations). John D. H. Stead has collaborated with scholars based in Canada, United States and United Kingdom. Frequent co-authors include Charles R. Neal, Stanley J. Watson, Huda Akil, Hymie Anisman, Fan Meng, Alec J. Jeffreys, Simon J. Evans, Opal A. McInnis, Robyn J. McQuaid and Sarah M. Clinton. Their work appears in journals such as PLoS ONE, Human Molecular Genetics, Electrophoresis, Scientific Reports and Cell Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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