James Peddy
Impact in
- Biological Psychiatry top 10%
- Tryptophan and brain disorders
- Physiology top 10%
- Alzheimer's disease research and treatments
- Calcium signaling and nucleotide metabolism
Papers in
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- Mitochondrial Function and Pathology 2
- Retinal Development and Disorders 2
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- Cellular transport and secretion 3
- Endoplasmic Reticulum Stress and Disease 1
- Co-authors
- Ralph A. Nixon (6 shared papers)Chris N. Goulbourne (6 shared papers)Ju‐Hyun Lee (3 shared papers)Philip Stavrides (6 shared papers)Monika Pawlik (3 shared papers)Cynthia Bleiwas (3 shared papers)Chunfeng Huo (4 shared papers)Martin J. Berg (3 shared papers)
- Journals
- eLife (2 papers)Autophagy (1 paper)Cell Reports (1 paper)Nature Neuroscience (1 paper)Journal of Neuroscience (1 paper)
- Partner nations
- United StatesGermany
In The Last Decade
James Peddy
5 papers receiving 519 citations
James Peddy's Hit Papers
Peers
Comparison fields: 5 of 69
- Biological Psychiatry 29
- Physiology 269
- Neurology 72
- Physiology 37
- Cell Biology 93
Countries citing papers authored by James Peddy
This map shows the geographic impact of James Peddy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James Peddy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James Peddy more than expected).
Fields of papers citing papers by James Peddy
This network shows the impact of papers produced by James Peddy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James Peddy. The network helps show where James Peddy may publish in the future.
Co-authors
The 25 scholars most cited alongside James Peddy, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Faulty autolysosome acidification in Alzheimer’s disease mouse models induces autophagic build-up of Aβ in neurons, yielding senile plaques Hit paper breakdown → | 2022 | 428 |
| 2 | 2020 | 74 | |
| 3 | 2022 | 19 | |
| 4 | 2025 | 1 | |
| 5 | 2025 | 1 | |
| 6 | 2025 | 0 |
About James Peddy
James Peddy is a scholar working on Molecular Biology, Cell Biology, Epidemiology, Physiology and Cellular and Molecular Neuroscience, having authored 6 papers that have together received 523 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (3 papers), Autophagy in Disease and Therapy (3 papers), Cellular transport and secretion (3 papers), Parkinson's Disease Mechanisms and Treatments (2 papers), Mitochondrial Function and Pathology (2 papers), Retinal Development and Disorders (2 papers), Genetic Neurodegenerative Diseases (2 papers) and Endoplasmic Reticulum Stress and Disease (1 paper). The work is most often cited by research in Biological Psychiatry (29 citations), Physiology (269 citations), Neurology (72 citations), Physiology (37 citations) and Cell Biology (93 citations). James Peddy has collaborated with scholars based in United States and Germany. Frequent co-authors include Ralph A. Nixon, Chris N. Goulbourne, Ju‐Hyun Lee, Philip Stavrides, Monika Pawlik, Cynthia Bleiwas, Chunfeng Huo, Martin J. Berg, Anna Pensalfini and Dun‐Sheng Yang. Their work appears in journals such as eLife, Autophagy, Cell Reports, Nature Neuroscience and Journal of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.