Iva Vukelić

581 citations
12 papers · 494 · h-index 8

Impact in

Papers in

    • Genomics, phytochemicals, and oxidative stress 5
    • FOXO transcription factor regulation 2
    • Extracellular vesicles in disease 2
    • Ubiquitin and proteasome pathways 1
    • Spinal Cord Injury Research 2
    • Chemotherapy-induced organ toxicity mitigation 1

Iva Vukelić

11 papers receiving 489 citations

Peers

Iva Vukelić
Comparison fields: 5 of 79
  • Biochemistry 28
  • Pharmacology 39
  • Molecular Biology 291
  • Oncology 89
  • Applied Microbiology and Biotechnology 6
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Junshan Ruan China
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Citations per field
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Citations per year

Countries citing papers authored by Iva Vukelić

Since Specialization
Citations

This map shows the geographic impact of Iva Vukelić's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Iva Vukelić with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Iva Vukelić more than expected).

Fields of papers citing papers by Iva Vukelić

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Iva Vukelić. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Iva Vukelić. The network helps show where Iva Vukelić may publish in the future.

Co-authors

The 25 scholars most cited alongside Iva Vukelić, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Iva Vukelić Line = papers co-authored together Iva Vukelić links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1 2012176
2 201878
3 202060
4 202058
5 201945
6 202229
7 201428
8 20248
9 20227
10
Nuclear factor erythroid 2-related factor 2 and choline acetyltransferase co-expression in rat spinal cord neurons after ischemia-reperfusion injury.
20194
11 20151
12 20260

About Iva Vukelić

Iva Vukelić is a scholar working on Molecular Biology, Pathology and Forensic Medicine, Oncology, Epidemiology and Immunology, having authored 12 papers that have together received 494 indexed citations. Recurring topics across this work include Genomics, phytochemicals, and oxidative stress (5 papers), FOXO transcription factor regulation (2 papers), Spinal Cord Injury Research (2 papers), MicroRNA in disease regulation (2 papers), Autophagy in Disease and Therapy (2 papers), Extracellular vesicles in disease (2 papers), Chemotherapy-induced organ toxicity mitigation (1 paper) and Ubiquitin and proteasome pathways (1 paper). The work is most often cited by research in Biochemistry (28 citations), Pharmacology (39 citations), Molecular Biology (291 citations), Oncology (89 citations) and Applied Microbiology and Biotechnology (6 citations). Iva Vukelić has collaborated with scholars based in Croatia, United States and Italy. Frequent co-authors include Iva Potočnjak, Robert Domitrović, Dijana Detel, Lara Batičić, Ines Oršolić, Maja Cokarić Brdovčak, Chen Katz, Slađana Bursać, Vedrana Filić and Kristina Grabušić. Their work appears in journals such as Food and Chemical Toxicology, Chemico-Biological Interactions, Free Radical Biology and Medicine, Diabetology & Metabolic Syndrome and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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