István Földi

824 citations
24 papers · 581 · h-index 15

Impact in

  • Aging top 10%
  • Cell Biology top 10%
    • Cellular Mechanics and Interactions
    • Microtubule and mitosis dynamics
    • Endoplasmic Reticulum Stress and Disease

Papers in

    • Signaling Pathways in Disease 2
    • Cellular Mechanics and Interactions 6
    • Microtubule and mitosis dynamics 4
    • Endoplasmic Reticulum Stress and Disease 2

István Földi

23 papers receiving 579 citations

Peers

István Földi
Comparison fields: 5 of 89
  • Aging 21
  • Cell Biology 158
  • Cellular and Molecular Neuroscience 149
  • Immunology 155
  • Biological Psychiatry 13
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Citations per field
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Citations per year

Countries citing papers authored by István Földi

Since Specialization
Citations

This map shows the geographic impact of István Földi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by István Földi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites István Földi more than expected).

Fields of papers citing papers by István Földi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by István Földi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by István Földi. The network helps show where István Földi may publish in the future.

Co-authors

The 25 scholars most cited alongside István Földi, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with István Földi Line = papers co-authored together István Földi links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 24 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2018157
2 201381
3 201738
4 201732
5 201928
6 200925
7 200824
8 201223
9 201121
10 201220
11 202017
12 201117
13 201817
14 201716
15 201916
16 201710
17 200710
18 20219
19 20179
20 20117

About István Földi

István Földi is a scholar working on Molecular Biology, Cell Biology, Cellular and Molecular Neuroscience, Physiology and Immunology, having authored 24 papers that have together received 581 indexed citations. Recurring topics across this work include Cellular Mechanics and Interactions (6 papers), Neurobiology and Insect Physiology Research (6 papers), Alzheimer's disease research and treatments (5 papers), Microtubule and mitosis dynamics (4 papers), Invertebrate Immune Response Mechanisms (2 papers), Signaling Pathways in Disease (2 papers), Nerve injury and regeneration (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). The work is most often cited by research in Aging (21 citations), Cell Biology (158 citations), Cellular and Molecular Neuroscience (149 citations), Immunology (155 citations) and Biological Psychiatry (13 citations). István Földi has collaborated with scholars based in Hungary, United Kingdom and Germany. Frequent co-authors include Alicia Hidalgo, Botond Penke, József Mihály, Tamás Janáky, Nicholas J. Gay, Jill S. Wentzell, Graham McIlroy, Janine Fenton, Mária Szűcs and Gábor Juhász. Their work appears in journals such as Development, The Journal of Cell Biology, PLoS Genetics, Cells and Electrophoresis.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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