Ingo Backert
Impact in
- Reproductive Medicine top 10%
- Sperm and Testicular Function
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- IL-33, ST2, and ILC Pathways
- Psoriasis: Treatment and Pathogenesis
- Immune Cell Function and Interaction
Papers in
- Genetics 4
- Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities 2
- Digestive system and related health 1
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- Epigenetics and DNA Methylation 1
- Protein Degradation and Inhibitors 1
- Co-authors
- Markus F. Neurath (4 shared papers)Clemens Neufert (3 shared papers)Stefan Wirtz (2 shared papers)Ulrike Mau (1 shared paper)Peter Kaiser (1 shared paper)Ludwig Kiesel (1 shared paper)Axel J. Hueber (1 shared paper)Michael Vieth (1 shared paper)
- Journals
- Human Reproduction (2 papers)The Journal of Immunology (1 paper)Gut (1 paper)Cell Reports (1 paper)Journal of Clinical Investigation (1 paper)
- Partner nations
- GermanyUnited StatesJapan
In The Last Decade
Ingo Backert
7 papers receiving 548 citations
Peers
Comparison fields: 5 of 66
- Reproductive Medicine 79
- Immunology 184
- Genetics 204
- Dermatology 36
- Oncology 95
Countries citing papers authored by Ingo Backert
This map shows the geographic impact of Ingo Backert's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ingo Backert with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ingo Backert more than expected).
Fields of papers citing papers by Ingo Backert
This network shows the impact of papers produced by Ingo Backert. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ingo Backert. The network helps show where Ingo Backert may publish in the future.
Co-authors
The 25 scholars most cited alongside Ingo Backert, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2016 | 147 | |
| 2 | 1998 | 100 | |
| 3 | 2013 | 98 | |
| 4 | 1997 | 75 | |
| 5 | 2014 | 66 | |
| 6 | 2014 | 56 | |
| 7 | 2016 | 22 |
About Ingo Backert
Ingo Backert is a scholar working on Genetics, Molecular Biology, Oncology, Immunology and Reproductive Medicine, having authored 7 papers that have together received 564 indexed citations. Recurring topics across this work include Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (2 papers), Cancer Cells and Metastasis (2 papers), Pharmacological Effects of Natural Compounds (1 paper), Epigenetics and DNA Methylation (1 paper), Digestive system and related health (1 paper), Protein Degradation and Inhibitors (1 paper), Sperm and Testicular Function (1 paper) and Psoriasis: Treatment and Pathogenesis (1 paper). The work is most often cited by research in Reproductive Medicine (79 citations), Immunology (184 citations), Genetics (204 citations), Dermatology (36 citations) and Oncology (95 citations). Ingo Backert has collaborated with scholars based in Germany, United States and Japan. Frequent co-authors include Markus F. Neurath, Clemens Neufert, Stefan Wirtz, Ulrike Mau, Peter Kaiser, Ludwig Kiesel, Axel J. Hueber, Michael Vieth, Hans Christian Probst and Kristina Scheibe. Their work appears in journals such as Human Reproduction, The Journal of Immunology, Gut, Cell Reports and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.