Idil Tuncali
Impact in
- Neurology top 10%
- Parkinson's Disease Mechanisms and Treatments
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- Lysosomal Storage Disorders Research
- Alzheimer's disease research and treatments
Papers in
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- Parkinson's Disease Mechanisms and Treatments 3
- Amyotrophic Lateral Sclerosis Research 1
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- Machine Learning in Bioinformatics 1
- Sphingolipid Metabolism and Signaling 1
- Bioinformatics and Genomic Networks 1
- Co-authors
- Clemens R. Scherzer (3 shared papers)Anne‐Marie Wills (2 shared papers)Yuliya I. Kuras (2 shared papers)Sebastian Virreira Winter (1 shared paper)Matthias Mann (1 shared paper)Kalpana Merchant (1 shared paper)Jiajie Peng (1 shared paper)Xuequn Shang (1 shared paper)
- Journals
- Briefings in Bioinformatics (1 paper)Scientific Reports (1 paper)Neurology (1 paper)npj Parkinson s Disease (1 paper)Cell Reports Medicine (1 paper)
- Partner nations
- United StatesChinaSouth Korea
In The Last Decade
Idil Tuncali
6 papers receiving 241 citations
Peers
Comparison fields: 5 of 73
- Neurology 97
- Neurology 23
- Physiology 69
- Cell Biology 37
- Computational Mathematics 1
Countries citing papers authored by Idil Tuncali
This map shows the geographic impact of Idil Tuncali's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Idil Tuncali with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Idil Tuncali more than expected).
Fields of papers citing papers by Idil Tuncali
This network shows the impact of papers produced by Idil Tuncali. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Idil Tuncali. The network helps show where Idil Tuncali may publish in the future.
Co-authors
The 25 scholars most cited alongside Idil Tuncali, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2022 | 103 | |
| 2 | 2020 | 61 | |
| 3 | 2020 | 34 | |
| 4 | 2021 | 31 | |
| 5 | 2023 | 8 | |
| 6 | 2023 | 5 |
About Idil Tuncali
Idil Tuncali is a scholar working on Neurology, Molecular Biology, Physiology, Pharmacology and Genetics, having authored 6 papers that have together received 242 indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (3 papers), Lysosomal Storage Disorders Research (2 papers), Machine Learning in Bioinformatics (1 paper), Cholinesterase and Neurodegenerative Diseases (1 paper), Sphingolipid Metabolism and Signaling (1 paper), Amyotrophic Lateral Sclerosis Research (1 paper), Bioinformatics and Genomic Networks (1 paper) and Child and Adolescent Psychosocial and Emotional Development (1 paper). The work is most often cited by research in Neurology (97 citations), Neurology (23 citations), Physiology (69 citations), Cell Biology (37 citations) and Computational Mathematics (1 citation). Idil Tuncali has collaborated with scholars based in United States, China and South Korea. Frequent co-authors include Clemens R. Scherzer, Anne‐Marie Wills, Yuliya I. Kuras, Sebastian Virreira Winter, Matthias Mann, Kalpana Merchant, Jiajie Peng, Xuequn Shang, Hansheng Xue and Özge Karayel. Their work appears in journals such as Briefings in Bioinformatics, Scientific Reports, Neurology, npj Parkinson s Disease and Cell Reports Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.