I. Imanaga
Impact in
- Sensory Systems top 5%
- Ion Channels and Receptors
-
- Neuroscience and Neuropharmacology Research
- Neuroscience and Neural Engineering
Papers in
-
- Ion channel regulation and function 21
- Connexins and lens biology 6
- Mitochondrial Function and Pathology 3
-
- Cardiac electrophysiology and arrhythmias 13
- Cardiovascular Function and Risk Factors 3
- Co-authors
- Masumi Inoue (16 shared papers)Naoji Fujishiro (7 shared papers)A. Uehara (4 shared papers)Narcis Tribulová (5 shared papers)Hiroshi Irisawa (1 shared paper)Masaki Kameyama (1 shared paper)Miyuki Nishi (1 shared paper)Hiroshi Takeshima (1 shared paper)
In The Last Decade
I. Imanaga
30 papers receiving 585 citations
Peers
Comparison fields: 5 of 66
- Sensory Systems 69
- Cellular and Molecular Neuroscience 188
- Cardiology and Cardiovascular Medicine 205
- Endocrine and Autonomic Systems 54
- Molecular Biology 432
Countries citing papers authored by I. Imanaga
This map shows the geographic impact of I. Imanaga's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I. Imanaga with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I. Imanaga more than expected).
Fields of papers citing papers by I. Imanaga
This network shows the impact of papers produced by I. Imanaga. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I. Imanaga. The network helps show where I. Imanaga may publish in the future.
Co-authors
The 25 scholars most cited alongside I. Imanaga, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 32 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2002 | 73 | |
| 2 | 1987 | 47 | |
| 3 | 1998 | 44 | |
| 4 | 2009 | 37 | |
| 5 | Thyroid hormones suppress epsilon-PKC signalling, down-regulate connexin-43 and increase lethal arrhythmia susceptibility in non-diabetic and diabetic rat hearts. | 2008 | 37 |
| 6 | 1993 | 34 | |
| 7 | 1996 | 34 | |
| 8 | 1995 | 29 | |
| 9 | 2000 | 29 | |
| 10 | 2002 | 26 | |
| 11 | Factors involved in the susceptibility of spontaneously hypertensive rats to low K+-induced arrhythmias. | 2003 | 23 |
| 12 | 1995 | 21 | |
| 13 | 1993 | 21 | |
| 14 | 1999 | 20 | |
| 15 | 1998 | 18 | |
| 16 | 1987 | 17 | |
| 17 | 2015 | 16 | |
| 18 | 1993 | 12 | |
| 19 | 1996 | 11 | |
| 20 | 1997 | 10 |
About I. Imanaga
I. Imanaga is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine, Cellular and Molecular Neuroscience, Pathology and Forensic Medicine and Clinical Biochemistry, having authored 32 papers that have together received 594 indexed citations. Recurring topics across this work include Ion channel regulation and function (21 papers), Cardiac electrophysiology and arrhythmias (13 papers), Neuroscience and Neuropharmacology Research (8 papers), Connexins and lens biology (6 papers), Neuroscience and Neural Engineering (5 papers), Mitochondrial Function and Pathology (3 papers), Cardiovascular Function and Risk Factors (3 papers) and Molecular Sensors and Ion Detection (2 papers). The work is most often cited by research in Sensory Systems (69 citations), Cellular and Molecular Neuroscience (188 citations), Cardiology and Cardiovascular Medicine (205 citations), Endocrine and Autonomic Systems (54 citations) and Molecular Biology (432 citations). I. Imanaga has collaborated with scholars based in Japan, Slovakia and Ukraine. Frequent co-authors include Masumi Inoue, Naoji Fujishiro, A. Uehara, Narcis Tribulová, Hiroshi Irisawa, Masaki Kameyama, Miyuki Nishi, Hiroshi Takeshima, Y. Sakamoto and Ľudmila Okruhlicová. Their work appears in journals such as American Journal of Physiology-Cell Physiology, The Journal of Physiology, Brain Research, Journal of Molecular and Cellular Cardiology and Physiological Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.