Hugh O. McDevitt
Impact in
- Immunology top 0.02%
- T-cell and B-cell Immunology
- Immune Cell Function and Interaction
- Immunotherapy and Immune Responses
- Immune Response and Inflammation
- Genetics top 0.05%
- Diabetes and associated disorders
Papers in
- Immunology 145
- T-cell and B-cell Immunology 101
- Immune Cell Function and Interaction 70
- Immunotherapy and Immune Responses 37
- Immune Response and Inflammation 20
- Genetics 77
- Diabetes and associated disorders 70
- Co-authors
- Roland Tisch (16 shared papers)Baruj Benacerraf (2 shared papers)John A. Todd (6 shared papers)Roland Liblau (14 shared papers)John I. Bell (3 shared papers)Chaim O. Jacob (7 shared papers)Steven M. Singer (8 shared papers)Gordon W. Duff (1 shared paper)
- Journals
- Proceedings of the National Academy of Sciences (37 papers)The Journal of Experimental Medicine (33 papers)The Journal of Immunology (18 papers)Immunogenetics (12 papers)Diabetes (7 papers)
- Partner nations
- United StatesUnited KingdomFrance
In The Last Decade
Hugh O. McDevitt
222 papers receiving 24.1k citations
Hugh O. McDevitt's Hit Papers
Peers
Comparison fields: 5 of 154
- Immunology 16.1k
- Genetics 7.8k
- Rheumatology 2.6k
- Radiology, Nuclear Medicine and Imaging 3.8k
- Endocrinology, Diabetes and Metabolism 2.4k
Countries citing papers authored by Hugh O. McDevitt
This map shows the geographic impact of Hugh O. McDevitt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hugh O. McDevitt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hugh O. McDevitt more than expected).
Fields of papers citing papers by Hugh O. McDevitt
This network shows the impact of papers produced by Hugh O. McDevitt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hugh O. McDevitt. The network helps show where Hugh O. McDevitt may publish in the future.
Co-authors
The 25 scholars most cited alongside Hugh O. McDevitt, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 224 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Effects of a polymorphism in the human tumor necrosis factor α promoter on transcriptional activation Hit paper breakdown → | 1997 | 1953 |
| 2 | HLA-DQβ gene contributes to susceptibility and resistance to insulin-dependent diabetes mellitus Hit paper breakdown → | 1987 | 1648 |
| 3 | In Vitro–expanded Antigen-specific Regulatory T Cells Suppress Autoimmune Diabetes Hit paper breakdown → | 2004 | 1022 |
| 4 | Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseases Hit paper breakdown → | 1995 | 944 |
| 5 | Insulin-Dependent Diabetes Mellitus Hit paper breakdown → | 1996 | 936 |
| 6 | Histocompatibility-Linked Immune Response Genes Hit paper breakdown → | 1972 | 927 |
| 7 | Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic mice Hit paper breakdown → | 1993 | 794 |
| 8 | A Molecular Basis for MHC Class II—Associated Autoimmunity Hit paper breakdown → | 1988 | 606 |
| 9 | Heritable major histocompatibility complex class II-associated differences in production of tumor necrosis factor alpha: relevance to genetic predisposition to systemic lupus erythematosus. Hit paper breakdown → | 1990 | 537 |
| 10 | Genetic Control of Specific Immune Responses Hit paper breakdown → | 1969 | 526 |
| 11 | Tumour necrosis factor-α in murine autoimmune 'lupus' nephritis Hit paper breakdown → | 1988 | 491 |
| 12 | 1990 | 371 | |
| 13 | 1989 | 346 | |
| 14 | 1987 | 328 | |
| 15 | 1987 | 326 | |
| 16 | 1988 | 310 | |
| 17 | 1965 | 308 | |
| 18 | 1971 | 305 | |
| 19 | Genetic Control of the Antibody Response: Relationship between Immune Response and Histocompatibility ( H-2 ) Type Hit paper breakdown → | 1969 | 298 |
| 20 | 1996 | 283 |
About Hugh O. McDevitt
Hugh O. McDevitt is a scholar working on Immunology, Genetics, Radiology, Nuclear Medicine and Imaging, Molecular Biology and Surgery, having authored 224 papers that have together received 25.8k indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (101 papers), Diabetes and associated disorders (70 papers), Immune Cell Function and Interaction (70 papers), Monoclonal and Polyclonal Antibodies Research (63 papers), Immunotherapy and Immune Responses (37 papers), Pancreatic function and diabetes (23 papers), Glycosylation and Glycoproteins Research (20 papers) and Immune Response and Inflammation (20 papers). The work is most often cited by research in Immunology (16.1k citations), Genetics (7.8k citations), Rheumatology (2.6k citations), Radiology, Nuclear Medicine and Imaging (3.8k citations) and Endocrinology, Diabetes and Metabolism (2.4k citations). Hugh O. McDevitt has collaborated with scholars based in United States, United Kingdom and France. Frequent co-authors include Roland Tisch, Baruj Benacerraf, John A. Todd, Roland Liblau, John I. Bell, Chaim O. Jacob, Steven M. Singer, Gordon W. Duff, Tarra L. McDowell and Julian Symons. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine, The Journal of Immunology, Immunogenetics and Diabetes.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.