Hsin‐Yi Tseng
Impact in
- Cell Biology top 10%
- Endoplasmic Reticulum Stress and Disease
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- Immunotherapy and Immune Responses
Papers in
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- Cell death mechanisms and regulation 5
- Melanoma and MAPK Pathways 5
- Histone Deacetylase Inhibitors Research 4
- Protein Degradation and Inhibitors 3
- Oncology 8
- Co-authors
- Xu Dong Zhang (15 shared papers)Chen Chen Jiang (12 shared papers)Lei Jin (12 shared papers)Kwang Hong Tay (9 shared papers)Amanda Croft (6 shared papers)Peter Hersey (13 shared papers)Su Guo (9 shared papers)Xu Guang Yan (6 shared papers)
In The Last Decade
Hsin‐Yi Tseng
29 papers receiving 817 citations
Peers
Comparison fields: 5 of 82
- Cell Biology 189
- Immunology 186
- Cancer Research 103
- Molecular Biology 472
- Epidemiology 192
Countries citing papers authored by Hsin‐Yi Tseng
This map shows the geographic impact of Hsin‐Yi Tseng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hsin‐Yi Tseng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hsin‐Yi Tseng more than expected).
Fields of papers citing papers by Hsin‐Yi Tseng
This network shows the impact of papers produced by Hsin‐Yi Tseng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hsin‐Yi Tseng. The network helps show where Hsin‐Yi Tseng may publish in the future.
Co-authors
The 25 scholars most cited alongside Hsin‐Yi Tseng, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 30 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 78 | |
| 2 | 2015 | 68 | |
| 3 | 2013 | 67 | |
| 4 | 2013 | 64 | |
| 5 | 2015 | 59 | |
| 6 | 2012 | 55 | |
| 7 | 2009 | 52 | |
| 8 | 2015 | 38 | |
| 9 | 2014 | 37 | |
| 10 | 2014 | 36 | |
| 11 | 2016 | 28 | |
| 12 | 2017 | 27 | |
| 13 | 2016 | 25 | |
| 14 | 2012 | 22 | |
| 15 | 2010 | 22 | |
| 16 | 2020 | 20 | |
| 17 | 2020 | 20 | |
| 18 | 2022 | 17 | |
| 19 | 2021 | 16 | |
| 20 | 2014 | 16 |
About Hsin‐Yi Tseng
Hsin‐Yi Tseng is a scholar working on Molecular Biology, Oncology, Epidemiology, Immunology and Cell Biology, having authored 30 papers that have together received 819 indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (7 papers), Cell death mechanisms and regulation (5 papers), Immunotherapy and Immune Responses (5 papers), Melanoma and MAPK Pathways (5 papers), Histone Deacetylase Inhibitors Research (4 papers), Endoplasmic Reticulum Stress and Disease (3 papers), Protein Degradation and Inhibitors (3 papers) and Impact of Technology on Adolescents (3 papers). The work is most often cited by research in Cell Biology (189 citations), Immunology (186 citations), Cancer Research (103 citations), Molecular Biology (472 citations) and Epidemiology (192 citations). Hsin‐Yi Tseng has collaborated with scholars based in Australia, China and Taiwan. Frequent co-authors include Xu Dong Zhang, Chen Chen Jiang, Lei Jin, Kwang Hong Tay, Amanda Croft, Peter Hersey, Su Guo, Xu Guang Yan, Qi Luan and Fritz Lai. Their work appears in journals such as Pigment Cell & Melanoma Research, Journal of Investigative Dermatology, Oncotarget, Autophagy and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.