Hideki Kaneda
Impact in
- Clinical Biochemistry top 1%
- Metabolism and Genetic Disorders
- Aging top 5%
Papers in
-
- Mitochondrial Function and Pathology 6
- Molecular Biology Techniques and Applications 3
- Genomics and Phylogenetic Studies 3
- CRISPR and Genetic Engineering 3
- Genetics 10
- Animal Genetics and Reproduction 6
- Co-authors
- Hiromichi Yonekawa (14 shared papers)J Hayashi (2 shared papers)Choji Taya (4 shared papers)K F Lindahl (1 shared paper)Jun‐Ichi Hayashi (3 shared papers)Hiroshi Shitara (4 shared papers)Yoichi Gondo (10 shared papers)Toshihiko Shiroishi (9 shared papers)
- Journals
- EXPERIMENTAL ANIMALS (4 papers)Genes & Genetic Systems (4 papers)Biochemical and Biophysical Research Communications (3 papers)Genetics (2 papers)Scientific Reports (2 papers)
- Partner nations
- JapanItalyUnited States
In The Last Decade
Hideki Kaneda
35 papers receiving 2.0k citations
Peers
Comparison fields: 5 of 113
- Clinical Biochemistry 310
- Aging 43
- Molecular Biology 1.4k
- Genetics 513
- Cellular and Molecular Neuroscience 271
Countries citing papers authored by Hideki Kaneda
This map shows the geographic impact of Hideki Kaneda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideki Kaneda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideki Kaneda more than expected).
Fields of papers citing papers by Hideki Kaneda
This network shows the impact of papers produced by Hideki Kaneda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideki Kaneda. The network helps show where Hideki Kaneda may publish in the future.
Co-authors
The 25 scholars most cited alongside Hideki Kaneda, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 37 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2007 | 440 | |
| 2 | 1995 | 390 | |
| 3 | 1998 | 193 | |
| 4 | 2000 | 128 | |
| 5 | 1994 | 104 | |
| 6 | 2001 | 100 | |
| 7 | 1998 | 92 | |
| 8 | 2005 | 79 | |
| 9 | 2005 | 65 | |
| 10 | 2007 | 50 | |
| 11 | 2001 | 45 | |
| 12 | 1998 | 38 | |
| 13 | 2010 | 28 | |
| 14 | 2017 | 28 | |
| 15 | 2014 | 27 | |
| 16 | 2002 | 26 | |
| 17 | 2000 | 25 | |
| 18 | 2016 | 24 | |
| 19 | 2009 | 24 | |
| 20 | A monogenic dominant mutation in Rom1 generated by N-ethyl-N-nitrosourea mutagenesis causes retinal degeneration in mice. | 2010 | 18 |
About Hideki Kaneda
Hideki Kaneda is a scholar working on Molecular Biology, Genetics, Cellular and Molecular Neuroscience, Public Health, Environmental and Occupational Health and Clinical Biochemistry, having authored 37 papers that have together received 2.1k indexed citations. Recurring topics across this work include Animal Genetics and Reproduction (6 papers), Mitochondrial Function and Pathology (6 papers), Reproductive Biology and Fertility (5 papers), Metabolism and Genetic Disorders (4 papers), Neurogenesis and neuroplasticity mechanisms (3 papers), Molecular Biology Techniques and Applications (3 papers), Genomics and Phylogenetic Studies (3 papers) and CRISPR and Genetic Engineering (3 papers). The work is most often cited by research in Clinical Biochemistry (310 citations), Aging (43 citations), Molecular Biology (1.4k citations), Genetics (513 citations) and Cellular and Molecular Neuroscience (271 citations). Hideki Kaneda has collaborated with scholars based in Japan, Italy and United States. Frequent co-authors include Hiromichi Yonekawa, J Hayashi, Choji Taya, K F Lindahl, Jun‐Ichi Hayashi, Hiroshi Shitara, Yoichi Gondo, Toshihiko Shiroishi, Yoshiyuki Sakuraba and Shigeharu Wakana. Their work appears in journals such as EXPERIMENTAL ANIMALS, Genes & Genetic Systems, Biochemical and Biophysical Research Communications, Genetics and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.