Heli Helander
Impact in
- Clinical Biochemistry top 5%
- Metabolism and Genetic Disorders
-
- Peroxisome Proliferator-Activated Receptors
- Mitochondrial Function and Pathology
- Microbial metabolism and enzyme function
- ATP Synthase and ATPases Research
Papers in
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- Peroxisome Proliferator-Activated Receptors 3
- Microbial Metabolic Engineering and Bioproduction 2
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- Metabolism and Genetic Disorders 4
- Co-authors
- J. Kalervo Hiltunen (4 shared papers)Werner Schmitz (3 shared papers)Ernst Conzelmann (3 shared papers)Raili Myllylä (1 shared paper)Minna Valtavaara (1 shared paper)Anna Maria Pirttilä (1 shared paper)Ari-Pekka Kvist (1 shared paper)Ulf Hellman (1 shared paper)
In The Last Decade
Heli Helander
14 papers receiving 418 citations
Peers
Comparison fields: 5 of 72
- Clinical Biochemistry 120
- Molecular Biology 309
- Cell Biology 54
- Cancer Research 49
- Genetics 84
Countries citing papers authored by Heli Helander
This map shows the geographic impact of Heli Helander's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heli Helander with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heli Helander more than expected).
Fields of papers citing papers by Heli Helander
This network shows the impact of papers produced by Heli Helander. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heli Helander. The network helps show where Heli Helander may publish in the future.
Co-authors
The 25 scholars most cited alongside Heli Helander, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1997 | 115 | |
| 2 | 1997 | 88 | |
| 3 | 2000 | 53 | |
| 4 | 2005 | 38 | |
| 5 | 2006 | 33 | |
| 6 | 1997 | 27 | |
| 7 | 1997 | 26 | |
| 8 | 2016 | 20 | |
| 9 | 2020 | 18 | |
| 10 | 2017 | 7 | |
| 11 | 2022 | 5 | |
| 12 | 2006 | 3 | |
| 13 | 2021 | 2 | |
| 14 | 2024 | 1 | |
| 15 | 2025 | 0 | |
| 16 | 2024 | 0 |
About Heli Helander
Heli Helander is a scholar working on Molecular Biology, Clinical Biochemistry, Genetics, Psychiatry and Mental health and Cellular and Molecular Neuroscience, having authored 16 papers that have together received 436 indexed citations. Recurring topics across this work include Metabolism and Genetic Disorders (4 papers), Peroxisome Proliferator-Activated Receptors (3 papers), Microbial Metabolic Engineering and Bioproduction (2 papers), Epilepsy research and treatment (2 papers), Genetic Neurodegenerative Diseases (2 papers), Genetics and Neurodevelopmental Disorders (2 papers), Folate and B Vitamins Research (2 papers) and Genomics and Rare Diseases (2 papers). The work is most often cited by research in Clinical Biochemistry (120 citations), Molecular Biology (309 citations), Cell Biology (54 citations), Cancer Research (49 citations) and Genetics (84 citations). Heli Helander has collaborated with scholars based in Finland, Germany and Sweden. Frequent co-authors include J. Kalervo Hiltunen, Werner Schmitz, Ernst Conzelmann, Raili Myllylä, Minna Valtavaara, Anna Maria Pirttilä, Ari-Pekka Kvist, Ulf Hellman, Matti Poutanen and Kari Majamaa. Their work appears in journals such as Journal of Biological Chemistry, Biochemical Journal, Neuropediatrics, Genomics and European Journal of Human Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.