Gary Kasof
Impact in
- Immunology top 10%
- interferon and immune responses
- Phagocytosis and Immune Regulation
- Molecular Biology top 10%
- Cell death mechanisms and regulation
- RNA Interference and Gene Delivery
- Ubiquitin and proteasome pathways
Papers in
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- Cell death mechanisms and regulation 7
-
- interferon and immune responses 2
- Co-authors
- Bruce Gomes (3 shared papers)Lakshmi Goyal (1 shared paper)Eileen White (2 shared papers)Derong Liu (2 shared papers)Matthew V. Lorenzi (2 shared papers)B S Speer (1 shared paper)Nicholas T. Ktistakis (1 shared paper)Jack‐Ansgar Bruun (1 shared paper)
- Journals
- Journal of Biological Chemistry (2 papers)Cell Discovery (1 paper)Cell Reports (1 paper)Oncogene (1 paper)Molecular and Cellular Biology (1 paper)
- Partner nations
- United StatesUnited KingdomNorway
In The Last Decade
Gary Kasof
11 papers receiving 953 citations
Peers
Comparison fields: 5 of 87
- Immunology 219
- Molecular Biology 699
- Cancer Research 130
- Physiology 34
- Epidemiology 178
Countries citing papers authored by Gary Kasof
This map shows the geographic impact of Gary Kasof's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gary Kasof with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gary Kasof more than expected).
Fields of papers citing papers by Gary Kasof
This network shows the impact of papers produced by Gary Kasof. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gary Kasof. The network helps show where Gary Kasof may publish in the future.
Co-authors
The 25 scholars most cited alongside Gary Kasof, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2001 | 385 | |
| 2 | 1999 | 176 | |
| 3 | 2019 | 110 | |
| 4 | 2000 | 102 | |
| 5 | 2001 | 83 | |
| 6 | 2020 | 59 | |
| 7 | 2025 | 17 | |
| 8 | 2014 | 16 | |
| 9 | 1996 | 14 | |
| 10 | 1995 | 12 | |
| 11 | 2010 | 1 |
About Gary Kasof
Gary Kasof is a scholar working on Molecular Biology, Immunology, Cancer Research, Cellular and Molecular Neuroscience and Oncology, having authored 11 papers that have together received 975 indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (7 papers), NF-κB Signaling Pathways (3 papers), Neuroscience and Neuropharmacology Research (2 papers), Cancer-related Molecular Pathways (2 papers), interferon and immune responses (2 papers), Nuclear Receptors and Signaling (2 papers), Autophagy in Disease and Therapy (2 papers) and Neurogenesis and neuroplasticity mechanisms (1 paper). The work is most often cited by research in Immunology (219 citations), Molecular Biology (699 citations), Cancer Research (130 citations), Physiology (34 citations) and Epidemiology (178 citations). Gary Kasof has collaborated with scholars based in United States, United Kingdom and Norway. Frequent co-authors include Bruce Gomes, Lakshmi Goyal, Eileen White, Derong Liu, Matthew V. Lorenzi, B S Speer, Nicholas T. Ktistakis, Jack‐Ansgar Bruun, Terje Johansen and Michal Mudd. Their work appears in journals such as Journal of Biological Chemistry, Cell Discovery, Cell Reports, Oncogene and Molecular and Cellular Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.