Frédéric Ezan

3.0k citations
36 papers · 2.6k · h-index 26

Impact in

Papers in

    • Melanoma and MAPK Pathways 4
    • DNA Repair Mechanisms 2
    • Pluripotent Stem Cells Research 2
    • Liver physiology and pathology 10

Frédéric Ezan

35 papers receiving 2.6k citations

Peers

Frédéric Ezan
Comparison fields: 5 of 118
  • Developmental Neuroscience 954
  • Cellular and Molecular Neuroscience 1.0k
  • Hepatology 220
  • Neurology 183
  • Cell Biology 327
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Jane E. Bottenstein United States
Clara Alfaro‐Cervelló Spain
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Fabienne Lamballe France
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Citations per field
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Citations per year

Countries citing papers authored by Frédéric Ezan

Since Specialization
Citations

This map shows the geographic impact of Frédéric Ezan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frédéric Ezan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frédéric Ezan more than expected).

Fields of papers citing papers by Frédéric Ezan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frédéric Ezan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frédéric Ezan. The network helps show where Frédéric Ezan may publish in the future.

Co-authors

The 25 scholars most cited alongside Frédéric Ezan, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Frédéric Ezan Line = papers co-authored together Frédéric Ezan links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 36 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1998473
2 1998435
3 1998173
4 1999158
5 2010154
6 202096
7 200793
8 199680
9 201077
10 200869
11 200468
12 201159
13 201259
14 200251
15 201746
16 201244
17 200541
18 202140
19 201238
20 200937

About Frédéric Ezan

Frédéric Ezan is a scholar working on Molecular Biology, Hepatology, Cellular and Molecular Neuroscience, Developmental Neuroscience and Cancer Research, having authored 36 papers that have together received 2.6k indexed citations. Recurring topics across this work include Liver physiology and pathology (10 papers), Neurogenesis and neuroplasticity mechanisms (6 papers), Axon Guidance and Neuronal Signaling (6 papers), Melanoma and MAPK Pathways (4 papers), Carcinogens and Genotoxicity Assessment (3 papers), 3D Printing in Biomedical Research (3 papers), DNA Repair Mechanisms (2 papers) and Pluripotent Stem Cells Research (2 papers). The work is most often cited by research in Developmental Neuroscience (954 citations), Cellular and Molecular Neuroscience (1.0k citations), Hepatology (220 citations), Neurology (183 citations) and Cell Biology (327 citations). Frédéric Ezan has collaborated with scholars based in France, United States and Spain. Frequent co-authors include Constantino Sotelo, Georges Baffet, Eduardo Soriano, Tom Curran, Gabriella D’Arcangelo, Luı́s de Lecea, Soledad Alcántara, Sophie Langouët, Christophe Frémin and Evelyne Bloch‐Gallego. Their work appears in journals such as Hepatology, Journal of Neuroscience, Journal of Cellular Physiology, Scientific Reports and Journal of Hepatology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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