Ewan E. Morrison

4.3k citations
68 papers · 3.3k · h-index 30

Impact in

Papers in

    • Ubiquitin and proteasome pathways 9
    • Wnt/β-catenin signaling in development and cancer 7
    • DNA Repair Mechanisms 4
    • Microtubule and mitosis dynamics 22

Ewan E. Morrison

67 papers receiving 3.2k citations

Peers

Ewan E. Morrison
Comparison fields: 5 of 123
  • Cell Biology 1.4k
  • Developmental Neuroscience 112
  • Molecular Biology 1.7k
  • Endocrine and Autonomic Systems 134
  • Genetics 542
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Li‐Yuan Yu‐Lee United States
Anna Castro France
Jan Sap United States
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Citations per field
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Citations per year

Countries citing papers authored by Ewan E. Morrison

Since Specialization
Citations

This map shows the geographic impact of Ewan E. Morrison's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ewan E. Morrison with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ewan E. Morrison more than expected).

Fields of papers citing papers by Ewan E. Morrison

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ewan E. Morrison. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ewan E. Morrison. The network helps show where Ewan E. Morrison may publish in the future.

Co-authors

The 25 scholars most cited alongside Ewan E. Morrison, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ewan E. Morrison Line = papers co-authored together Ewan E. Morrison links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 68 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2005423
2 1998192
3 2008181
4 1997171
5 2002161
6 2008146
7 1998132
8 2009122
9 2000111
10 2005109
11 201096
12 200590
13 200777
14 199869
15 201165
16 200263
17 200457
18 200355
19 201455
20 200754

About Ewan E. Morrison

Ewan E. Morrison is a scholar working on Molecular Biology, Cell Biology, Genetics, Oncology and Epidemiology, having authored 68 papers that have together received 3.3k indexed citations. Recurring topics across this work include Microtubule and mitosis dynamics (22 papers), Ubiquitin and proteasome pathways (9 papers), Wnt/β-catenin signaling in development and cancer (7 papers), Virus-based gene therapy research (7 papers), Herpesvirus Infections and Treatments (6 papers), Stress Responses and Cortisol (5 papers), Adrenal Hormones and Disorders (5 papers) and DNA Repair Mechanisms (4 papers). The work is most often cited by research in Cell Biology (1.4k citations), Developmental Neuroscience (112 citations), Molecular Biology (1.7k citations), Endocrine and Autonomic Systems (134 citations) and Genetics (542 citations). Ewan E. Morrison has collaborated with scholars based in United Kingdom, United States and Slovakia. Frequent co-authors include Jon M. Askham, María G. Castro, D M Meredith, Jacquelyn Bond, Holly V. Goodson, David M. Meredith, Yi‐Fen Wang, Kevin T. Vaughan, Michelle Peckham and A.F. Markham. Their work appears in journals such as PLoS ONE, British Journal of Cancer, BMC Cell Biology, Oncogene and Biochemical Society Transactions.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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