Edward Seto

30.4k citations
175 papers · 25.2k · 12 hit papers · h-index 75

Impact in

    • Sirtuins and Resveratrol in Medicine
    • Histone Deacetylase Inhibitors Research
    • Epigenetics and DNA Methylation
    • Protein Degradation and Inhibitors
    • Ubiquitin and proteasome pathways
    • Genomics and Chromatin Dynamics
    • Cancer-related gene regulation

Papers in

    • Histone Deacetylase Inhibitors Research 105
    • Protein Degradation and Inhibitors 49
    • Epigenetics and DNA Methylation 39
    • Ubiquitin and proteasome pathways 31
    • Genomics and Chromatin Dynamics 19
    • Signaling Pathways in Disease 11
    • Peptidase Inhibition and Analysis 20

Edward Seto

174 papers receiving 24.7k citations

Edward Seto's Hit Papers

HDACs and HDAC Inhibitors in Cancer Development and Therapy 2016 · 999 citations
9990+11+23Years since publication4008001.2k

Peers

Edward Seto
Comparison fields: 5 of 143
  • Geriatrics and Gerontology 1.3k
  • Molecular Biology 19.9k
  • Oncology 4.9k
  • Cancer Research 1.9k
  • Immunology 2.5k
Replace Saadi Khochbin with:
Saadi Khochbin France
Chunaram Choudhary Denmark
Adi Kimchi Israel
Junjie Chen United States
Bernhard Lüscher Germany
Ronen Marmorstein United States
Yoshihide Tsujimoto Japan
Saverio Minucci Italy
René H. Medema Netherlands
J. Alan Diehl United States
Edward Seto relative to Saadi Khochbin France Saadi Khochbin's profile →
Citations per field
00.5×3.0×
Saadi Khochbin · 1×
Citations per year

Countries citing papers authored by Edward Seto

Since Specialization
Citations

This map shows the geographic impact of Edward Seto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward Seto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward Seto more than expected).

Fields of papers citing papers by Edward Seto

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edward Seto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward Seto. The network helps show where Edward Seto may publish in the future.

Co-authors

The 25 scholars most cited alongside Edward Seto, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Edward Seto Line = papers co-authored together Edward Seto links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 175 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Acetylation and deacetylation of non-histone proteins
Hit paper breakdown →
20051362
2
A complex containing N-CoR, mSln3 and histone deacetylase mediates transcriptional repression
Hit paper breakdown →
19971101
3
The Rpd3/Hda1 family of lysine deacetylases: from bacteria and yeast to mice and men
Hit paper breakdown →
20081016
4
HDACs and HDAC Inhibitors in Cancer Development and Therapy
Hit paper breakdown →
2016999
5
Transcriptional repression by YY1, a human GLI-Krüippel-related protein, and relief of repression by adenovirus E1A protein
Hit paper breakdown →
1991944
6
Histone deacetylases and cancer
Hit paper breakdown →
2007867
7
HATs and HDACs: from structure, function and regulation to novel strategies for therapy and prevention
Hit paper breakdown →
2007848
8
Lysine Acetylation: Codified Crosstalk with Other Posttranslational Modifications
Hit paper breakdown →
2008845
9
Histone Deacetylases Associated with the mSin3 Corepressor Mediate Mad Transcriptional Repression
Hit paper breakdown →
1997840
10
Inhibition of Histone Deacetylase 6 Acetylates and Disrupts the Chaperone Function of Heat Shock Protein 90
Hit paper breakdown →
2005666
11
HDAC6 Modulates Cell Motility by Altering the Acetylation Level of Cortactin
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2007608
12
Histone deacetylases, transcriptional control, and cancer
Hit paper breakdown →
2000545
13 1996492
14 1992470
15 2000435
16 1991424
17 1999423
18 1997394
19 2001388
20 2008371

About Edward Seto

Edward Seto is a scholar working on Molecular Biology, Oncology, Genetics, Immunology and Epidemiology, having authored 175 papers that have together received 25.2k indexed citations. Recurring topics across this work include Histone Deacetylase Inhibitors Research (105 papers), Protein Degradation and Inhibitors (49 papers), Epigenetics and DNA Methylation (39 papers), Ubiquitin and proteasome pathways (31 papers), Peptidase Inhibition and Analysis (20 papers), Genomics and Chromatin Dynamics (19 papers), Sirtuins and Resveratrol in Medicine (13 papers) and Signaling Pathways in Disease (11 papers). The work is most often cited by research in Geriatrics and Gerontology (1.3k citations), Molecular Biology (19.9k citations), Oncology (4.9k citations), Cancer Research (1.9k citations) and Immunology (2.5k citations). Edward Seto has collaborated with scholars based in United States, Canada and China. Frequent co-authors include Michele A. Glozak, Wen‐Ming Yang, Xiang‐Jiao Yang, Nilanjan Sengupta, Yixuan Li, Thomas Shenk, Xiang-Jiao Yang, Xiaohong Zhang, X-J Yang and James Davie. Their work appears in journals such as Journal of Biological Chemistry, Molecular and Cellular Biology, Blood, Proceedings of the National Academy of Sciences and Oncogene.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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