E. Reit

494 citations
15 papers · 459 · h-index 8

Impact in

Papers in

E. Reit

15 papers receiving 366 citations

Peers

E. Reit
Comparison fields: 5 of 66
  • Cellular and Molecular Neuroscience 161
  • Cardiology and Cardiovascular Medicine 163
  • Genetics 62
  • Endocrine and Autonomic Systems 34
  • Physiology 83
Replace P. Szerdahelyi with:
P. Szerdahelyi Hungary
M. D. Galva Italy
Seiko Tsuda Japan
Else M�ller-Schweinitzer Germany
Yu-Ting Xuan United States
Joseph R. Stimers United States
Caroline Lagneux France
Hiroyasu Fukuta Japan
Anjaneyulu S. Tadepalli United States
M. Fujiwara Japan
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Citations per field
00.5×6.2×
P. Szerdahelyi · 1×
Citations per year

Countries citing papers authored by E. Reit

Since Specialization
Citations

This map shows the geographic impact of E. Reit's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. Reit with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. Reit more than expected).

Fields of papers citing papers by E. Reit

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. Reit. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. Reit. The network helps show where E. Reit may publish in the future.

Co-authors

The 9 scholars most cited alongside E. Reit, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with E. Reit Line = papers co-authored together E. Reit links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 1965175
2 196669
3
Actions of angiotensin on the adrenal medulla and autonomic ganglia.
197258
4 196850
5 196639
6 196925
7 197110
8 19738
9 19676
10 19714
11 19754
12 19664
13 19733
14 19693
15 19771

About E. Reit

E. Reit is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Cardiology and Cardiovascular Medicine, Pharmacology and Oncology, having authored 15 papers that have together received 459 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (4 papers), Neuropeptides and Animal Physiology (4 papers), Cardiovascular and exercise physiology (2 papers), Epilepsy research and treatment (2 papers), Neuroscience and Neuropharmacology Research (2 papers), Heart Rate Variability and Autonomic Control (2 papers), Drug Transport and Resistance Mechanisms (2 papers) and Antibiotics Pharmacokinetics and Efficacy (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (161 citations), Cardiology and Cardiovascular Medicine (163 citations), Genetics (62 citations), Endocrine and Autonomic Systems (34 citations) and Physiology (83 citations). E. Reit has collaborated with scholars based in United States, Tanzania and United Kingdom. Frequent co-authors include G P Lewis, James W. Aiken, Geoffrey Hall, Peter Cervoni, A. Jay Block, Nicholas J. Giarman, Allan P. Gray, W. Feldberg and O Hudlická. Their work appears in journals such as Journal of Pharmacology and Experimental Therapeutics, British Journal of Pharmacology, The Journal of Physiology, European Journal of Pharmacology and Journal of Medicinal Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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