David J. MacEwan

9.0k citations
93 papers · 4.5k · h-index 38

Impact in

Papers in

    • Protein Kinase Regulation and GTPase Signaling 19
    • Receptor Mechanisms and Signaling 13
    • Cell death mechanisms and regulation 13
    • Genomics, phytochemicals, and oxidative stress 10
    • Immune Response and Inflammation 10

David J. MacEwan

92 papers receiving 4.4k citations

Peers

David J. MacEwan
Comparison fields: 5 of 114
  • Cancer Research 627
  • Immunology 875
  • Molecular Biology 2.4k
  • Hematology 371
  • Cellular and Molecular Neuroscience 606
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Citations per field
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Citations per year

Countries citing papers authored by David J. MacEwan

Since Specialization
Citations

This map shows the geographic impact of David J. MacEwan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David J. MacEwan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David J. MacEwan more than expected).

Fields of papers citing papers by David J. MacEwan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David J. MacEwan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David J. MacEwan. The network helps show where David J. MacEwan may publish in the future.

Co-authors

The 25 scholars most cited alongside David J. MacEwan, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David J. MacEwan Line = papers co-authored together David J. MacEwan links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 93 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2002493
2 2002328
3 2012250
4 2008184
5 2007171
6 2001150
7 2001147
8 2002144
9 2017123
10 2008115
11 201386
12 199983
13 200382
14 200275
15 201275
16 199675
17 201170
18 201169
19 201168
20 199963

About David J. MacEwan

David J. MacEwan is a scholar working on Molecular Biology, Immunology, Cancer Research, Cellular and Molecular Neuroscience and Oncology, having authored 93 papers that have together received 4.5k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (19 papers), Receptor Mechanisms and Signaling (13 papers), Cell death mechanisms and regulation (13 papers), NF-κB Signaling Pathways (13 papers), Immune Response and Inflammation (10 papers), Genomics, phytochemicals, and oxidative stress (10 papers), Chronic Lymphocytic Leukemia Research (9 papers) and Neuroscience and Neuropharmacology Research (8 papers). The work is most often cited by research in Cancer Research (627 citations), Immunology (875 citations), Molecular Biology (2.4k citations), Hematology (371 citations) and Cellular and Molecular Neuroscience (606 citations). David J. MacEwan has collaborated with scholars based in United Kingdom, Belgium and United States. Frequent co-authors include Stuart A. Rushworth, Kristian M. Bowles, Lyubov Zaitseva, Megan Y. Murray, Graeme Milligan, Peter Vandenabeele, Michelle Connell, Shona M. McFarlane, Niraj Shah and Maria A. O’Connell. Their work appears in journals such as FEBS Letters, Biochemical Society Transactions, Cell Cycle, Biochemical Journal and Blood.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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