David Batt

1.6k citations
21 papers · 1.3k · h-index 14

Impact in

  • Oncology top 10%
    • Cancer-related Molecular Pathways
    • HER2/EGFR in Cancer Research
    • PI3K/AKT/mTOR signaling in cancer
    • Protein Kinase Regulation and GTPase Signaling
    • Melanoma and MAPK Pathways

Papers in

    • Melanoma and MAPK Pathways 5
    • Protein Kinase Regulation and GTPase Signaling 2
    • Polyomavirus and related diseases 3
    • HER2/EGFR in Cancer Research 2
    • Cancer-related Molecular Pathways 2

David Batt

21 papers receiving 1.3k citations

Peers

David Batt
Comparison fields: 5 of 87
  • Oncology 411
  • Molecular Biology 944
  • Cancer Research 166
  • Pathology and Forensic Medicine 148
  • Genetics 80
Replace Yusuke Nakamura with:
Yusuke Nakamura Japan
Patrick Kwok‐Shing Ng United States
A.‐C. Andres Switzerland
Jung-Hsin Hsu United States
Wanleng Deng United States
Melody Stallings‐Mann United States
Helena Jernberg‐Wiklund Sweden
Eftihia Cocolakis Canada
Tracey Rowlands United States
Daniel S. Liscia United States
David Batt relative to Yusuke Nakamura Japan Yusuke Nakamura's profile →
Citations per field
00.5×1.6×
Yusuke Nakamura · 1×
Citations per year

Countries citing papers authored by David Batt

Since Specialization
Citations

This map shows the geographic impact of David Batt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Batt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Batt more than expected).

Fields of papers citing papers by David Batt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Batt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Batt. The network helps show where David Batt may publish in the future.

Co-authors

The 25 scholars most cited alongside David Batt, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David Batt Line = papers co-authored together David Batt links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 21 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1999492
2 2010178
3 2006113
4 1999106
5 200846
6
B-Raf kinase inhibitors for cancer treatment.
200744
7 200743
8 199842
9 200641
10 199439
11 200732
12 199923
13 199423
14 199917
15 201010
16 20059
17 20088
18 19945
19 19954
20 20154

About David Batt

David Batt is a scholar working on Molecular Biology, Oncology, Pathology and Forensic Medicine, Organic Chemistry and Genetics, having authored 21 papers that have together received 1.3k indexed citations. Recurring topics across this work include Melanoma and MAPK Pathways (5 papers), Cancer Mechanisms and Therapy (5 papers), Synthesis and biological activity (3 papers), Polyomavirus and related diseases (3 papers), Plant Virus Research Studies (2 papers), HER2/EGFR in Cancer Research (2 papers), Cancer-related Molecular Pathways (2 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). The work is most often cited by research in Oncology (411 citations), Molecular Biology (944 citations), Cancer Research (166 citations), Pathology and Forensic Medicine (148 citations) and Genetics (80 citations). David Batt has collaborated with scholars based in United States, Switzerland and Germany. Frequent co-authors include Thomas M. Roberts, Noriaki Nakamura, William R. Sellers, Francisca Vázquez, Shivapriya Ramaswamy, Gordon Carmichael, Naeem Yusuff, Nedret Altıok, Markus Warmuth and Hava Avraham. Their work appears in journals such as Molecular Cancer Therapeutics, Journal of Biological Chemistry, Cancer Cell, Proceedings of the National Academy of Sciences and Cancer Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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