David Baltimore

182.3k citations
805 papers · 148.4k · 68 hit papers · h-index 194

Impact in

  • Virology top 0.01%
    • HIV Research and Treatment
  • Immunology top 0.01%
    • Immune Cell Function and Interaction
    • T-cell and B-cell Immunology
    • Immune Response and Inflammation

Papers in

    • Immune Cell Function and Interaction 81
    • T-cell and B-cell Immunology 80
    • NF-κB Signaling Pathways 79

David Baltimore

786 papers receiving 139.2k citations

David Baltimore's Hit Papers

30 Years of NF-κB: A Blossoming of Relevance to Human Pathobiology 2017 · 1.6k citations
1.6k0+6+13Years since publication50010001.5k

Peers

David Baltimore
Comparison fields: 5 of 212
  • Virology 12.0k
  • Immunology 46.6k
  • Cancer Research 30.5k
  • Molecular Biology 75.1k
  • Hematology 10.0k
Replace Tom Maniatis with:
Tom Maniatis United States
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G. Kroemer France
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Citations per field
00.5×2×4×5.2×
Tom Maniatis · 1×
Citations per year

Countries citing papers authored by David Baltimore

Since Specialization
Citations

This map shows the geographic impact of David Baltimore's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Baltimore with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Baltimore more than expected).

Fields of papers citing papers by David Baltimore

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Baltimore. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Baltimore. The network helps show where David Baltimore may publish in the future.

Co-authors

The 25 scholars most cited alongside David Baltimore, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David Baltimore Line = papers co-authored together David Baltimore links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 805 papers — load more, or switch the sort, to bring in the rest.

#Work
1
NF-κB-dependent induction of microRNA miR-146, an inhibitor targeted to signaling proteins of innate immune responses
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20063766
2
An Essential Role for NF-κB in Preventing TNF-α-Induced Cell Death
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19962831
3
NF-κB: Ten Years After
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19962808
4
Multiple nuclear factors interact with the immunoglobulin enhancer sequences
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19862401
5
A new DNA binding and dimerization motif in immunoglobulin enhancer binding, daughterless, MyoD, and myc proteins
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19892341
6
Production of high-titer helper-free retroviruses by transient transfection.
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19932163
7
IκB: a Specific Inhibitor of the NF-κB Transcription Factor
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19881981
8
An inducible transcription factor activates expression of human immunodeficiency virus in T cells
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19871878
9
Induction of Chronic Myelogenous Leukemia in Mice by the P210 bcr/abl Gene of the Philadelphia Chromosome
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19901856
10
Inducibility of κ immunoglobulin enhancer-binding protein NF-κB by a posttranslational mechanism
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19861785
11
Germline Transmission and Tissue-Specific Expression of Transgenes Delivered by Lentiviral Vectors
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20021685
12
Construction of a retrovirus packaging mutant and its use to produce helper-free defective retrovirus
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19831669
13
Viral RNA-dependent DNA Polymerase: RNA-dependent DNA Polymerase in Virions of RNA Tumour Viruses
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19701629
14
MicroRNA-155 is induced during the macrophage inflammatory response
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20071624
15
Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-κB
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19951603
16
30 Years of NF-κB: A Blossoming of Relevance to Human Pathobiology
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20171572
17
Interactions between heterologous helix-loop-helix proteins generate complexes that bind specifically to a common DNA sequence
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19891568
18
The IκB-NF-κB Signaling Module: Temporal Control and Selective Gene Activation
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20021530
19
NF-κB: A pleiotropic mediator of inducible and tissue-specific gene control
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19891481
20
The “initiator” as a transcription control element
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19891366

About David Baltimore

David Baltimore is a scholar working on Immunology, Cancer Research, Virology, Molecular Biology and Infectious Diseases, having authored 805 papers that have together received 148.4k indexed citations. Recurring topics across this work include Virus-based gene therapy research (99 papers), Viral Infections and Immunology Research (98 papers), Immune Cell Function and Interaction (81 papers), T-cell and B-cell Immunology (80 papers), NF-κB Signaling Pathways (79 papers), Monoclonal and Polyclonal Antibodies Research (78 papers), HIV Research and Treatment (74 papers) and RNA and protein synthesis mechanisms (67 papers). The work is most often cited by research in Virology (12.0k citations), Immunology (46.6k citations), Cancer Research (30.5k citations), Molecular Biology (75.1k citations) and Hematology (10.0k citations). David Baltimore has collaborated with scholars based in United States, Canada and Germany. Frequent co-authors include Patrick A. Baeuerle, Ranjan Sen, Mark Boldin, Konstantin D. Taganov, Amer A. Beg, Ryan M. O’Connell, Michael J. Lenardo, Dinesh S. Rao, Martin Scott and Cornelis Murre. Their work appears in journals such as Proceedings of the National Academy of Sciences, Cell, Journal of Virology, Molecular and Cellular Biology and Science.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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