Daniela Stadel
Impact in
- Physiology top 2%
- Calcium signaling and nucleotide metabolism
- Lysosomal Storage Disorders Research
- Cell Biology top 5%
- Endoplasmic Reticulum Stress and Disease
- Cellular transport and secretion
Papers in
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- Cellular Mechanics and Interactions 2
- Hippo pathway signaling and YAP/TAZ 2
- Endoplasmic Reticulum Stress and Disease 1
- Cellular transport and secretion 1
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- Autophagy in Disease and Therapy 3
- Co-authors
- Christian Behrends (4 shared papers)Miep Helfrich (1 shared paper)H. Suzuki (1 shared paper)Sagar Bhogaraju (1 shared paper)Karthik Maddi (1 shared paper)Philippe Pierre (1 shared paper)Doris Popovic (1 shared paper)Fraser P. Coxon (1 shared paper)
- Journals
- Molecular Cell (2 papers)Current Biology (1 paper)Biochimica et Biophysica Acta (BBA) - Molecular Cell Research (1 paper)
- Partner nations
- GermanyUnited KingdomFrance
In The Last Decade
Daniela Stadel
4 papers receiving 798 citations
Daniela Stadel's Hit Papers
Peers
Comparison fields: 5 of 73
- Physiology 147
- Cell Biology 332
- Epidemiology 480
- Aging 13
- Parasitology 41
Countries citing papers authored by Daniela Stadel
This map shows the geographic impact of Daniela Stadel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniela Stadel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniela Stadel more than expected).
Fields of papers citing papers by Daniela Stadel
This network shows the impact of papers produced by Daniela Stadel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniela Stadel. The network helps show where Daniela Stadel may publish in the future.
Co-authors
The 25 scholars most cited alongside Daniela Stadel, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | PLEKHM1 Regulates Autophagosome-Lysosome Fusion through HOPS Complex and LC3/GABARAP Proteins Hit paper breakdown → | 2014 | 451 |
| 2 | 2013 | 236 | |
| 3 | 2015 | 98 | |
| 4 | 2019 | 21 |
About Daniela Stadel
Daniela Stadel is a scholar working on Cell Biology, Epidemiology, Molecular Biology, Cellular and Molecular Neuroscience and Physiology, having authored 4 papers that have together received 806 indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (3 papers), Cellular Mechanics and Interactions (2 papers), Hippo pathway signaling and YAP/TAZ (2 papers), Heat shock proteins research (1 paper), Endoplasmic Reticulum Stress and Disease (1 paper), Hereditary Neurological Disorders (1 paper), Lysosomal Storage Disorders Research (1 paper) and Cellular transport and secretion (1 paper). The work is most often cited by research in Physiology (147 citations), Cell Biology (332 citations), Epidemiology (480 citations), Aging (13 citations) and Parasitology (41 citations). Daniela Stadel has collaborated with scholars based in Germany, United Kingdom and France. Frequent co-authors include Christian Behrends, Miep Helfrich, H. Suzuki, Sagar Bhogaraju, Karthik Maddi, Philippe Pierre, Doris Popovic, Fraser P. Coxon, Ivan Đikić and Andrea Gubaš. Their work appears in journals such as Molecular Cell, Current Biology and Biochimica et Biophysica Acta (BBA) - Molecular Cell Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.