Anna Ulbricht
Impact in
- Cell Biology top 10%
- Endoplasmic Reticulum Stress and Disease
- Cellular Mechanics and Interactions
- Aging top 10%
Papers in
-
- Heat shock proteins research 2
- Ubiquitin and proteasome pathways 1
- Mitochondrial Function and Pathology 1
-
- Autophagy in Disease and Therapy 4
- Co-authors
- Jörg Höhfeld (6 shared papers)Sebastian Gehlert (2 shared papers)Wilhelm Bloch (2 shared papers)Thorsten Schiffer (1 shared paper)Rudolf Volkmer (2 shared papers)Dieter O. Fürst (2 shared papers)Victor Tapia (2 shared papers)Waldemar Kolanus (1 shared paper)
- Journals
- Autophagy (2 papers)Communicative & Integrative Biology (1 paper)Biochimica et Biophysica Acta (BBA) - Molecular Cell Research (1 paper)Molecular Biology of the Cell (1 paper)Current Biology (1 paper)
- Partner nations
- GermanyUkraineSwitzerland
In The Last Decade
Anna Ulbricht
7 papers receiving 591 citations
Peers
Comparison fields: 5 of 72
- Cell Biology 207
- Aging 19
- Rehabilitation 41
- Molecular Biology 377
- Epidemiology 156
Countries citing papers authored by Anna Ulbricht
This map shows the geographic impact of Anna Ulbricht's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anna Ulbricht with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anna Ulbricht more than expected).
Fields of papers citing papers by Anna Ulbricht
This network shows the impact of papers produced by Anna Ulbricht. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anna Ulbricht. The network helps show where Anna Ulbricht may publish in the future.
Co-authors
The 25 scholars most cited alongside Anna Ulbricht, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 236 | |
| 2 | 2015 | 136 | |
| 3 | 2016 | 55 | |
| 4 | 2016 | 54 | |
| 5 | 2013 | 44 | |
| 6 | 2013 | 44 | |
| 7 | 2014 | 25 |
About Anna Ulbricht
Anna Ulbricht is a scholar working on Molecular Biology, Epidemiology, Cell Biology, Physiology and Infectious Diseases, having authored 7 papers that have together received 594 indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (4 papers), Endoplasmic Reticulum Stress and Disease (3 papers), Adipose Tissue and Metabolism (2 papers), Heat shock proteins research (2 papers), Cellular Mechanics and Interactions (2 papers), Cellular transport and secretion (2 papers), Ubiquitin and proteasome pathways (1 paper) and Mitochondrial Function and Pathology (1 paper). The work is most often cited by research in Cell Biology (207 citations), Aging (19 citations), Rehabilitation (41 citations), Molecular Biology (377 citations) and Epidemiology (156 citations). Anna Ulbricht has collaborated with scholars based in Germany, Ukraine and Switzerland. Frequent co-authors include Jörg Höhfeld, Sebastian Gehlert, Wilhelm Bloch, Thorsten Schiffer, Rudolf Volkmer, Dieter O. Fürst, Victor Tapia, Waldemar Kolanus, Padmanabhan Vakeel and Christian Behrends. Their work appears in journals such as Autophagy, Communicative & Integrative Biology, Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, Molecular Biology of the Cell and Current Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.