Daniel E. Foxler
Impact in
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- Cancer, Hypoxia, and Metabolism
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
Papers in
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- Cancer, Hypoxia, and Metabolism 3
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- RNA modifications and cancer 7
- Congenital heart defects research 2
- RNA Research and Splicing 2
- Pluripotent Stem Cells Research 1
- Co-authors
- Tyson V. Sharp (8 shared papers)Gregory D. Longmore (3 shared papers)Victoria H. James (3 shared papers)Yunfeng Feng (2 shared papers)Katherine S. Bridge (2 shared papers)Maureen Mee (2 shared papers)Dimitris Lagos (2 shared papers)Jóhannes Björnsson (1 shared paper)
- Journals
- Proceedings of the National Academy of Sciences (2 papers)Oncotarget (1 paper)Stem Cell Research (1 paper)Cell Reports (1 paper)FEBS Letters (1 paper)
- Partner nations
- United KingdomUnited StatesTaiwan
In The Last Decade
Daniel E. Foxler
9 papers receiving 334 citations
Peers
Comparison fields: 5 of 53
- Cancer Research 124
- Aging 7
- Cell Biology 52
- Molecular Biology 214
- Oncology 47
Countries citing papers authored by Daniel E. Foxler
This map shows the geographic impact of Daniel E. Foxler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel E. Foxler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel E. Foxler more than expected).
Fields of papers citing papers by Daniel E. Foxler
This network shows the impact of papers produced by Daniel E. Foxler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel E. Foxler. The network helps show where Daniel E. Foxler may publish in the future.
Co-authors
The 25 scholars most cited alongside Daniel E. Foxler, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2012 | 83 | |
| 2 | 2010 | 57 | |
| 3 | 2017 | 56 | |
| 4 | 2008 | 49 | |
| 5 | 2016 | 27 | |
| 6 | 2018 | 21 | |
| 7 | 2017 | 18 | |
| 8 | 2011 | 16 | |
| 9 | 2011 | 12 |
About Daniel E. Foxler
Daniel E. Foxler is a scholar working on Cancer Research, Molecular Biology, Surgery, Physiology and Oncology, having authored 9 papers that have together received 339 indexed citations. Recurring topics across this work include RNA modifications and cancer (7 papers), Cancer, Hypoxia, and Metabolism (3 papers), Congenital Diaphragmatic Hernia Studies (2 papers), Congenital heart defects research (2 papers), RNA Research and Splicing (2 papers), Pluripotent Stem Cells Research (1 paper), Histiocytic Disorders and Treatments (1 paper) and Adipose Tissue and Metabolism (1 paper). The work is most often cited by research in Cancer Research (124 citations), Aging (7 citations), Cell Biology (52 citations), Molecular Biology (214 citations) and Oncology (47 citations). Daniel E. Foxler has collaborated with scholars based in United Kingdom, United States and Taiwan. Frequent co-authors include Tyson V. Sharp, Gregory D. Longmore, Victoria H. James, Yunfeng Feng, Katherine S. Bridge, Maureen Mee, Dimitris Lagos, Jóhannes Björnsson, Sigurður Ingvarsson and Dumitru Constantin‐Teodosiu. Their work appears in journals such as Proceedings of the National Academy of Sciences, Oncotarget, Stem Cell Research, Cell Reports and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.