D. Izard
Impact in
- Endocrinology top 1%
- Enterobacteriaceae and Cronobacter Research
- Molecular Medicine top 1%
- Antibiotic Resistance in Bacteria
Papers in
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- Genomics and Phylogenetic Studies 30
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- Enterobacteriaceae and Cronobacter Research 27
- Co-authors
- H Leclerc (61 shared papers)F. Gavini (41 shared papers)K. Kersters (6 shared papers)J. De Ley (5 shared papers)N. Baida (9 shared papers)M. Elomari (9 shared papers)Joris Mergaert (4 shared papers)Fouad Dabboussi (12 shared papers)
In The Last Decade
D. Izard
91 papers receiving 2.3k citations
Peers
Comparison fields: 5 of 120
- Endocrinology 467
- Molecular Medicine 365
- Plant Science 821
- Food Science 349
- Ecology 449
Countries citing papers authored by D. Izard
This map shows the geographic impact of D. Izard's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D. Izard with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D. Izard more than expected).
Fields of papers citing papers by D. Izard
This network shows the impact of papers produced by D. Izard. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D. Izard. The network helps show where D. Izard may publish in the future.
Co-authors
The 25 scholars most cited alongside D. Izard, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 103 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 1989 | 309 | |
| 2 | 2002 | 173 | |
| 3 | 2015 | 150 | |
| 4 | 1996 | 102 | |
| 5 | 1991 | 91 | |
| 6 | 1981 | 88 | |
| 7 | 1979 | 87 | |
| 8 | 1997 | 85 | |
| 9 | 1999 | 73 | |
| 10 | 1999 | 72 | |
| 11 | 2002 | 61 | |
| 12 | 1987 | 59 | |
| 13 | 2003 | 56 | |
| 14 | 1988 | 56 | |
| 15 | 1983 | 55 | |
| 16 | 1981 | 50 | |
| 17 | 1996 | 46 | |
| 18 | 1999 | 42 | |
| 19 | 2011 | 41 | |
| 20 | 1999 | 40 |
About D. Izard
D. Izard is a scholar working on Molecular Biology, Endocrinology, Molecular Medicine, Ecology and Pharmacology, having authored 103 papers that have together received 2.5k indexed citations. Recurring topics across this work include Antibiotic Resistance in Bacteria (31 papers), Genomics and Phylogenetic Studies (30 papers), Enterobacteriaceae and Cronobacter Research (27 papers), Microbial Community Ecology and Physiology (21 papers), Antibiotics Pharmacokinetics and Efficacy (17 papers), Plant Pathogenic Bacteria Studies (14 papers), Probiotics and Fermented Foods (10 papers) and Antibiotic Use and Resistance (8 papers). The work is most often cited by research in Endocrinology (467 citations), Molecular Medicine (365 citations), Plant Science (821 citations), Food Science (349 citations) and Ecology (449 citations). D. Izard has collaborated with scholars based in France, Belgium and Lebanon. Frequent co-authors include H Leclerc, F. Gavini, K. Kersters, J. De Ley, N. Baida, M. Elomari, Joris Mergaert, Fouad Dabboussi, Christine Mielcarek and P.A. Trinel. Their work appears in journals such as Journal of Clinical Microbiology, INTERNATIONAL JOURNAL OF SYSTEMATIC AND EVOLUTIONARY MICROBIOLOGY, Systematic and Applied Microbiology, Canadian Journal of Microbiology and Research in Microbiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.