Chun‐Xia Wan

414 citations
10 papers · 350 · h-index 10

Impact in

Papers in

    • Bioactive Natural Diterpenoids Research 1
    • Cell death mechanisms and regulation 1
    • Autophagy in Disease and Therapy 4

Chun‐Xia Wan

10 papers receiving 348 citations

Peers

Chun‐Xia Wan
Comparison fields: 5 of 70
  • Pharmacology 29
  • Pharmacology 42
  • Cardiology and Cardiovascular Medicine 50
  • Molecular Biology 164
  • Epidemiology 71
Replace Lu Xu with:
Lu Xu China
Huogen Lu Canada
Eleonora Derlindati Italy
Shengban You China
Raeesa A. Mohamad Saudi Arabia
Zi-Yue Wan China
Xiaojun Bai China
Xin Fang China
Meizhu Wu China
Chun‐Xia Wan relative to Lu Xu China Lu Xu's profile →
Citations per field
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Citations per year

Countries citing papers authored by Chun‐Xia Wan

Since Specialization
Citations

This map shows the geographic impact of Chun‐Xia Wan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chun‐Xia Wan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chun‐Xia Wan more than expected).

Fields of papers citing papers by Chun‐Xia Wan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chun‐Xia Wan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chun‐Xia Wan. The network helps show where Chun‐Xia Wan may publish in the future.

Co-authors

The 25 scholars most cited alongside Chun‐Xia Wan, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Chun‐Xia Wan Line = papers co-authored together Chun‐Xia Wan links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 201970
2 201951
3 201849
4 202036
5 201731
6 201829
7 201928
8 202124
9 201719
10
Tetrandrine down-regulates expression of miRNA-155 to inhibit signal-induced NF-κB activation in a rat model of diabetes mellitus.
201513

About Chun‐Xia Wan

Chun‐Xia Wan is a scholar working on Molecular Biology, Epidemiology, Pharmacology, Cardiology and Cardiovascular Medicine and Cancer Research, having authored 10 papers that have together received 350 indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (4 papers), Flavonoids in Medical Research (2 papers), Bioactive Natural Diterpenoids Research (1 paper), NF-κB Signaling Pathways (1 paper), Membrane Separation Technologies (1 paper), Berberine and alkaloids research (1 paper), Cell death mechanisms and regulation (1 paper) and Peptidase Inhibition and Analysis (1 paper). The work is most often cited by research in Pharmacology (29 citations), Pharmacology (42 citations), Cardiology and Cardiovascular Medicine (50 citations), Molecular Biology (164 citations) and Epidemiology (71 citations). Chun‐Xia Wan has collaborated with scholars based in China and United States. Frequent co-authors include Qizhu Tang, Man Xu, Wei Deng, Qingqing Wu, Di Fan, Huibo Wang, Hai‐Ming Wu, Libo Liu, Zhen‐Guo Ma and Ning Zhang. Their work appears in journals such as Cell Death and Disease, Journal of Pharmacological Sciences, Journal of Cellular Physiology, International Journal of Molecular Medicine and Molecular Nutrition & Food Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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