Chris Kay
Impact in
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- Genetic Neurodegenerative Diseases
- Neurology top 2%
- Neurological disorders and treatments
Papers in
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- Mitochondrial Function and Pathology 15
- Photosynthetic Processes and Mechanisms 11
- Muscle Physiology and Disorders 5
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- Genetic Neurodegenerative Diseases 24
- Co-authors
- Michael R. Hayden (25 shared papers)Blair R. Leavitt (2 shared papers)Christopher A. Ross (2 shared papers)Rachael I. Scahill (1 shared paper)Edward J. Wild (1 shared paper)Sarah J. Tabrizi (1 shared paper)Martha Nance (1 shared paper)Gillian P. Bates (1 shared paper)
- Journals
- Biochemistry (6 papers)Biochemical Journal (5 papers)Journal of Biological Chemistry (4 papers)Genetics in Medicine (3 papers)FEBS Letters (3 papers)
- Partner nations
- United StatesUnited KingdomCanada
In The Last Decade
Chris Kay
68 papers receiving 3.0k citations
Chris Kay's Hit Papers
Peers
Comparison fields: 5 of 133
- Cellular and Molecular Neuroscience 1.5k
- Neurology 495
- Molecular Biology 1.7k
- Biological Psychiatry 35
- Aging 27
Countries citing papers authored by Chris Kay
This map shows the geographic impact of Chris Kay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chris Kay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chris Kay more than expected).
Fields of papers citing papers by Chris Kay
This network shows the impact of papers produced by Chris Kay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chris Kay. The network helps show where Chris Kay may publish in the future.
Co-authors
The 25 scholars most cited alongside Chris Kay, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 70 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Huntington disease Hit paper breakdown → | 2015 | 1162 |
| 2 | 2000 | 139 | |
| 3 | 2019 | 135 | |
| 4 | 2015 | 117 | |
| 5 | 1991 | 81 | |
| 6 | 2016 | 78 | |
| 7 | 2006 | 74 | |
| 8 | 2015 | 70 | |
| 9 | 2016 | 69 | |
| 10 | 2013 | 61 | |
| 11 | 2018 | 57 | |
| 12 | 2017 | 49 | |
| 13 | 2013 | 46 | |
| 14 | 2014 | 44 | |
| 15 | 1988 | 36 | |
| 16 | 2020 | 36 | |
| 17 | 2013 | 35 | |
| 18 | 1986 | 35 | |
| 19 | 2018 | 34 | |
| 20 | 1985 | 33 |
About Chris Kay
Chris Kay is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Epidemiology, Public Health, Environmental and Occupational Health and Cell Biology, having authored 70 papers that have together received 3.0k indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (24 papers), Mitochondrial Function and Pathology (15 papers), Photosynthetic Processes and Mechanisms (11 papers), Trypanosoma species research and implications (9 papers), Insect symbiosis and bacterial influences (6 papers), Neurological disorders and treatments (5 papers), Muscle Physiology and Disorders (5 papers) and Research on Leishmaniasis Studies (5 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.5k citations), Neurology (495 citations), Molecular Biology (1.7k citations), Biological Psychiatry (35 citations) and Aging (27 citations). Chris Kay has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Michael R. Hayden, Blair R. Leavitt, Christopher A. Ross, Rachael I. Scahill, Edward J. Wild, Sarah J. Tabrizi, Martha Nance, Gillian P. Bates, James F. Gusella and Ronald Wetzel. Their work appears in journals such as Biochemistry, Biochemical Journal, Journal of Biological Chemistry, Genetics in Medicine and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.