Robert T. Abraham
Impact in
- Cancer Research top 0.1%
- Cancer, Hypoxia, and Metabolism
- Molecular Biology top 0.05%
- PI3K/AKT/mTOR signaling in cancer
- DNA Repair Mechanisms
- Protein Kinase Regulation and GTPase Signaling
Papers in
-
- PI3K/AKT/mTOR signaling in cancer 48
- Protein Kinase Regulation and GTPase Signaling 30
- DNA Repair Mechanisms 21
- Immunology 70
- Immune Cell Function and Interaction 32
- T-cell and B-cell Immunology 29
- Co-authors
- Gary G. Chiang (11 shared papers)Larry M. Karnitz (18 shared papers)Gregory J. Wiederrecht (4 shared papers)Randal S. Tibbetts (6 shared papers)Jann N. Sarkaria (5 shared papers)Shubha Bagrodia (2 shared papers)Benjamin D. Hopkins (1 shared paper)Lewis C. Cantley (1 shared paper)
- Journals
- Journal of Biological Chemistry (26 papers)The Journal of Immunology (16 papers)Molecular and Cellular Biology (14 papers)Journal of Medicinal Chemistry (7 papers)Molecular Cancer Therapeutics (6 papers)
- Partner nations
- United StatesCanadaUnited Kingdom
In The Last Decade
Robert T. Abraham
216 papers receiving 28.7k citations
Robert T. Abraham's Hit Papers
Peers
Comparison fields: 5 of 166
- Cancer Research 4.1k
- Molecular Biology 19.4k
- Immunology 5.7k
- Oncology 6.9k
- Cell Biology 3.1k
Countries citing papers authored by Robert T. Abraham
This map shows the geographic impact of Robert T. Abraham's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert T. Abraham with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert T. Abraham more than expected).
Fields of papers citing papers by Robert T. Abraham
This network shows the impact of papers produced by Robert T. Abraham. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert T. Abraham. The network helps show where Robert T. Abraham may publish in the future.
Co-authors
The 25 scholars most cited alongside Robert T. Abraham, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 216 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | The PI3K Pathway in Human Disease Hit paper breakdown → | 2017 | 1973 |
| 2 | Cell cycle checkpoint signaling through the ATM and ATR kinases Hit paper breakdown → | 2001 | 1620 |
| 3 | Regulation of 4E-BP1 phosphorylation: a novel two-step mechanism Hit paper breakdown → | 1999 | 1074 |
| 4 | Regulation of Hypoxia-Inducible Factor 1α Expression and Function by the Mammalian Target of Rapamycin Hit paper breakdown → | 2002 | 983 |
| 5 | Inhibition of ATM and ATR kinase activities by the radiosensitizing agent, caffeine. Hit paper breakdown → | 1999 | 914 |
| 6 | A role for ATR in the DNA damage-induced phosphorylation of p53 Hit paper breakdown → | 1999 | 853 |
| 7 | Phosphorylation of the Translational Repressor PHAS-I by the Mammalian Target of Rapamycin Hit paper breakdown → | 1997 | 808 |
| 8 | Isolation of a Protein Target of the FKBP12-Rapamycin Complex in Mammalian Cells Hit paper breakdown → | 1995 | 716 |
| 9 | Direct inhibition of the signaling functions of the mammalian target of rapamycin by the phosphoinositide 3‐kinase inhibitors, wortmannin and LY294002. Hit paper breakdown → | 1996 | 635 |
| 10 | Wortmannin, a potent and selective inhibitor of phosphatidylinositol-3-kinase. Hit paper breakdown → | 1994 | 612 |
| 11 | A direct linkage between the phosphoinositide 3-kinase-AKT signaling pathway and the mammalian target of rapamycin in mitogen-stimulated and transformed cells. Hit paper breakdown → | 2000 | 609 |
| 12 | IMMUNOPHARMACOLOGY OF RAPAMYCIN Hit paper breakdown → | 1996 | 537 |
| 13 | Inhibition of phosphoinositide 3-kinase related kinases by the radiosensitizing agent wortmannin. | 1998 | 473 |
| 14 | 2005 | 463 | |
| 15 | 2004 | 447 | |
| 16 | 2000 | 407 | |
| 17 | 2004 | 361 | |
| 18 | 2005 | 338 | |
| 19 | 2002 | 332 | |
| 20 | 2015 | 304 |
About Robert T. Abraham
Robert T. Abraham is a scholar working on Molecular Biology, Immunology, Oncology, Cancer Research and Cell Biology, having authored 216 papers that have together received 29.2k indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (48 papers), Immune Cell Function and Interaction (32 papers), Protein Kinase Regulation and GTPase Signaling (30 papers), T-cell and B-cell Immunology (29 papers), DNA Repair Mechanisms (21 papers), Cytokine Signaling Pathways and Interactions (18 papers), Cancer-related Molecular Pathways (17 papers) and Microtubule and mitosis dynamics (14 papers). The work is most often cited by research in Cancer Research (4.1k citations), Molecular Biology (19.4k citations), Immunology (5.7k citations), Oncology (6.9k citations) and Cell Biology (3.1k citations). Robert T. Abraham has collaborated with scholars based in United States, Canada and United Kingdom. Frequent co-authors include Gary G. Chiang, Larry M. Karnitz, Gregory J. Wiederrecht, Randal S. Tibbetts, Jann N. Sarkaria, Shubha Bagrodia, Benjamin D. Hopkins, Lewis C. Cantley, Honyin Chiu and Christine C. Hudson. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Immunology, Molecular and Cellular Biology, Journal of Medicinal Chemistry and Molecular Cancer Therapeutics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.