Beth E. Drees

492 citations
9 papers · 436 · h-index 7

Impact in

    • Cell Adhesion Molecules Research
    • Cellular Mechanics and Interactions
    • Cellular transport and secretion
    • Hippo pathway signaling and YAP/TAZ
    • Microtubule and mitosis dynamics

Papers in

    • Cellular Mechanics and Interactions 3
    • Cellular transport and secretion 2
    • Chronic Lymphocytic Leukemia Research 2

Beth E. Drees

9 papers receiving 425 citations

Peers

Beth E. Drees
Comparison fields: 5 of 59
  • Immunology and Allergy 91
  • Cell Biology 236
  • Molecular Biology 261
  • Genetics 30
  • Biophysics 14
Replace Malika Boukhelifa with:
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Heli Suila Finland
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Taiko Sukezane Japan
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Citations per field
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Citations per year

Countries citing papers authored by Beth E. Drees

Since Specialization
Citations

This map shows the geographic impact of Beth E. Drees's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beth E. Drees with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beth E. Drees more than expected).

Fields of papers citing papers by Beth E. Drees

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beth E. Drees. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beth E. Drees. The network helps show where Beth E. Drees may publish in the future.

Co-authors

The 25 scholars most cited alongside Beth E. Drees, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Beth E. Drees Line = papers co-authored together Beth E. Drees links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 2000147
2 1999102
3 200758
4 200835
5 200335
6 200434
7 200220
8 20014
9 20041

About Beth E. Drees

Beth E. Drees is a scholar working on Cell Biology, Genetics, Molecular Biology, Cellular and Molecular Neuroscience and Radiology, Nuclear Medicine and Imaging, having authored 9 papers that have together received 436 indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (6 papers), Protein Kinase Regulation and GTPase Signaling (4 papers), Cellular Mechanics and Interactions (3 papers), Cellular transport and secretion (2 papers), Chronic Lymphocytic Leukemia Research (2 papers), Axon Guidance and Neuronal Signaling (1 paper), Protein Degradation and Inhibitors (1 paper) and Monoclonal and Polyclonal Antibodies Research (1 paper). The work is most often cited by research in Immunology and Allergy (91 citations), Cell Biology (236 citations), Molecular Biology (261 citations), Genetics (30 citations) and Biophysics (14 citations). Beth E. Drees has collaborated with scholars based in United States, Switzerland and Germany. Frequent co-authors include Mary C. Beckerle, Evelyne Friederich, Roy M. Golsteyn, Daniel Louvard, Glenn D. Prestwich, Paul O. Neilsen, Robert J. Eddy, Shu-Chin Yip, John S. Condeelis and Jonathan Backer. Their work appears in journals such as Expert Opinion on Therapeutic Patents, Journal of Cell Science, Journal of Biological Chemistry, Biochemical Journal and Combinatorial Chemistry & High Throughput Screening.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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