Beth A. Stevens
Impact in
- Neurology top 0.01%
- Neuroinflammation and Neurodegeneration Mechanisms
- Biological Psychiatry top 0.02%
- Tryptophan and brain disorders
Papers in
- Neurology 83
- Neuroinflammation and Neurodegeneration Mechanisms 79
-
- Neurogenesis and neuroplasticity mechanisms 28
- Co-authors
- Ben A. Barres (13 shared papers)Michael W. Salter (1 shared paper)Dorothy P. Schafer (10 shared papers)Emily K. Lehrman (8 shared papers)Soyon Hong (9 shared papers)Lasse Dissing‐Olesen (8 shared papers)R. Douglas Fields (9 shared papers)Timothy R. Hammond (6 shared papers)
- Journals
- Cell (8 papers)Nature Communications (8 papers)Neuron (8 papers)Journal of Neuroscience (8 papers)Science (5 papers)
- Partner nations
- United StatesUnited KingdomCanada
In The Last Decade
Beth A. Stevens
162 papers receiving 36.2k citations
Beth A. Stevens's Hit Papers
Peers
Comparison fields: 5 of 190
- Neurology 19.3k
- Biological Psychiatry 4.0k
- Developmental Neuroscience 4.7k
- Cellular and Molecular Neuroscience 7.9k
- Behavioral Neuroscience 1.3k
Countries citing papers authored by Beth A. Stevens
This map shows the geographic impact of Beth A. Stevens's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beth A. Stevens with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beth A. Stevens more than expected).
Fields of papers citing papers by Beth A. Stevens
This network shows the impact of papers produced by Beth A. Stevens. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beth A. Stevens. The network helps show where Beth A. Stevens may publish in the future.
Co-authors
The 25 scholars most cited alongside Beth A. Stevens, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 168 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Neurotoxic reactive astrocytes are induced by activated microglia Hit paper breakdown → | 2017 | 6019 |
| 2 | Microglia Sculpt Postnatal Neural Circuits in an Activity and Complement-Dependent Manner Hit paper breakdown → | 2012 | 3212 |
| 3 | The Classical Complement Cascade Mediates CNS Synapse Elimination Hit paper breakdown → | 2007 | 2601 |
| 4 | Complement and microglia mediate early synapse loss in Alzheimer mouse models Hit paper breakdown → | 2016 | 2543 |
| 5 | Schizophrenia risk from complex variation of complement component 4 Hit paper breakdown → | 2016 | 1826 |
| 6 | Single-Cell RNA Sequencing of Microglia throughout the Mouse Lifespan and in the Injured Brain Reveals Complex Cell-State Changes Hit paper breakdown → | 2018 | 1592 |
| 7 | Microglia emerge as central players in brain disease Hit paper breakdown → | 2017 | 1359 |
| 8 | Microglia states and nomenclature: A field at its crossroads Hit paper breakdown → | 2022 | 1302 |
| 9 | The Complement System: An Unexpected Role in Synaptic Pruning During Development and Disease Hit paper breakdown → | 2012 | 891 |
| 10 | The Role of Microglia in the Healthy Brain: Figure 1. Hit paper breakdown → | 2011 | 849 |
| 11 | 2006 | 564 | |
| 12 | Microglia: Dynamic Mediators of Synapse Development and Plasticity Hit paper breakdown → | 2015 | 553 |
| 13 | A complement–microglial axis drives synapse loss during virus-induced memory impairment Hit paper breakdown → | 2016 | 524 |
| 14 | Complement C3 deficiency protects against neurodegeneration in aged plaque-rich APP/PS1 mice Hit paper breakdown → | 2017 | 484 |
| 15 | 2002 | 473 | |
| 16 | 2012 | 465 | |
| 17 | 2015 | 446 | |
| 18 | 2011 | 390 | |
| 19 | 2018 | 370 | |
| 20 | Local externalization of phosphatidylserine mediates developmental synaptic pruning by microglia Hit paper breakdown → | 2020 | 316 |
About Beth A. Stevens
Beth A. Stevens is a scholar working on Neurology, Developmental Neuroscience, Cellular and Molecular Neuroscience, Immunology and Physiology, having authored 168 papers that have together received 36.8k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (79 papers), Neuroscience and Neuropharmacology Research (31 papers), Neurogenesis and neuroplasticity mechanisms (28 papers), Alzheimer's disease research and treatments (25 papers), Immune cells in cancer (22 papers), Tryptophan and brain disorders (8 papers), Nerve injury and regeneration (8 papers) and Complement system in diseases (7 papers). The work is most often cited by research in Neurology (19.3k citations), Biological Psychiatry (4.0k citations), Developmental Neuroscience (4.7k citations), Cellular and Molecular Neuroscience (7.9k citations) and Behavioral Neuroscience (1.3k citations). Beth A. Stevens has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Ben A. Barres, Michael W. Salter, Dorothy P. Schafer, Emily K. Lehrman, Soyon Hong, Lasse Dissing‐Olesen, R. Douglas Fields, Timothy R. Hammond, Alan R. Mardinly and Amanda G. Kautzman. Their work appears in journals such as Cell, Nature Communications, Neuron, Journal of Neuroscience and Science.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.