Benito Casanova

885 citations
16 papers · 751 · h-index 13

Impact in

Papers in

    • Metabolism, Diabetes, and Cancer 7
    • Cell death mechanisms and regulation 4
    • Chronic Lymphocytic Leukemia Research 6

Benito Casanova

16 papers receiving 732 citations

Peers

Benito Casanova
Comparison fields: 5 of 80
  • Genetics 190
  • Immunology and Allergy 102
  • Complementary and Manual Therapy 23
  • Nephrology 65
  • Nutrition and Dietetics 121
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Citations per field
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Citations per year

Countries citing papers authored by Benito Casanova

Since Specialization
Citations

This map shows the geographic impact of Benito Casanova's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benito Casanova with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benito Casanova more than expected).

Fields of papers citing papers by Benito Casanova

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benito Casanova. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benito Casanova. The network helps show where Benito Casanova may publish in the future.

Co-authors

The 22 scholars most cited alongside Benito Casanova, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Benito Casanova Line = papers co-authored together Benito Casanova links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 1995137
2 1999119
3
A critical role for ras-mediated, epidermal growth factor receptor-dependent angiogenesis in mouse skin carcinogenesis.
200296
4
Engagement of alpha4beta1 integrin by fibronectin induces in vitro resistance of B chronic lymphocytic leukemia cells to fludarabine.
200279
5 200271
6 200041
7 199734
8 200332
9 200431
10 200328
11 200128
12 199619
13 199614
14 199312
15 19958
16 19982

About Benito Casanova

Benito Casanova is a scholar working on Molecular Biology, Genetics, Oncology, Pathology and Forensic Medicine and Surgery, having authored 16 papers that have together received 751 indexed citations. Recurring topics across this work include Metabolism, Diabetes, and Cancer (7 papers), Chronic Lymphocytic Leukemia Research (6 papers), Cancer-related Molecular Pathways (4 papers), Cell death mechanisms and regulation (4 papers), Lymphoma Diagnosis and Treatment (2 papers), Cell Adhesion Molecules Research (2 papers), Parathyroid Disorders and Treatments (2 papers) and Adipose Tissue and Metabolism (2 papers). The work is most often cited by research in Genetics (190 citations), Immunology and Allergy (102 citations), Complementary and Manual Therapy (23 citations), Nephrology (65 citations) and Nutrition and Dietetics (121 citations). Benito Casanova has collaborated with scholars based in Spain and Germany. Frequent co-authors include Ángeles García-Pardo, Mercedes Garcı́a-Gila, Adela Rovira, Francisco Arrieta, José A. García‐Marco, Augusto Silva, Laura Sanz, José V. Moyano, M. T. de la Fuente and Claudio J. Conti. Their work appears in journals such as Diabetologia, Biochemical and Biophysical Research Communications, Leukemia, Metabolism and Molecular Biology of the Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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