Bastian Baumgartner
Impact in
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- Antioxidant Activity and Oxidative Stress
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- NF-κB Signaling Pathways
Papers in
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- Immune Response and Inflammation 2
- Atherosclerosis and Cardiovascular Diseases 1
- IL-33, ST2, and ILC Pathways 1
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- Cell death mechanisms and regulation 1
- Muscle Physiology and Disorders 1
- RNA regulation and disease 1
- Co-authors
- Korbinian Brand (4 shared papers)D. Neumeier (4 shared papers)Sharon Page (4 shared papers)Günther Loidl (2 shared papers)Marianne Hayn (1 shared paper)H. W. Löms Ziegler‐Heitbrock (1 shared paper)Claudia Fischer (1 shared paper)Cornelius Fischer (2 shared papers)
- Journals
- Leukemia (1 paper)Atherosclerosis (1 paper)The Cerebellum (1 paper)Journal of Biological Chemistry (1 paper)Toxicology and Applied Pharmacology (1 paper)
- Partner nations
- GermanySwitzerlandTunisia
In The Last Decade
Bastian Baumgartner
5 papers receiving 260 citations
Peers
Comparison fields: 5 of 63
- Biochemistry 36
- Cancer Research 71
- Hematology 36
- Immunology 61
- Clinical Biochemistry 15
Countries citing papers authored by Bastian Baumgartner
This map shows the geographic impact of Bastian Baumgartner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bastian Baumgartner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bastian Baumgartner more than expected).
Fields of papers citing papers by Bastian Baumgartner
This network shows the impact of papers produced by Bastian Baumgartner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bastian Baumgartner. The network helps show where Bastian Baumgartner may publish in the future.
Co-authors
The 25 scholars most cited alongside Bastian Baumgartner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1999 | 139 | |
| 2 | 2002 | 71 | |
| 3 | 2013 | 38 | |
| 4 | 1999 | 11 | |
| 5 | 1999 | 4 |
About Bastian Baumgartner
Bastian Baumgartner is a scholar working on Immunology, Molecular Biology, Cancer Research, Cellular and Molecular Neuroscience and Rheumatology, having authored 5 papers that have together received 263 indexed citations. Recurring topics across this work include NF-κB Signaling Pathways (2 papers), Immune Response and Inflammation (2 papers), Atherosclerosis and Cardiovascular Diseases (1 paper), Cell death mechanisms and regulation (1 paper), Muscle Physiology and Disorders (1 paper), IL-33, ST2, and ILC Pathways (1 paper), Genetic Neurodegenerative Diseases (1 paper) and RNA regulation and disease (1 paper). The work is most often cited by research in Biochemistry (36 citations), Cancer Research (71 citations), Hematology (36 citations), Immunology (61 citations) and Clinical Biochemistry (15 citations). Bastian Baumgartner has collaborated with scholars based in Germany, Switzerland and Tunisia. Frequent co-authors include Korbinian Brand, D. Neumeier, Sharon Page, Günther Loidl, Marianne Hayn, H. W. Löms Ziegler‐Heitbrock, Claudia Fischer, Cornelius Fischer, Christoph Schmid and C. von Schilling. Their work appears in journals such as Leukemia, Atherosclerosis, The Cerebellum, Journal of Biological Chemistry and Toxicology and Applied Pharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.