Bart De Vil

483 citations
7 papers · 224 · h-index 7

Impact in

    • Traumatic Brain Injury and Neurovascular Disturbances
    • Amyotrophic Lateral Sclerosis Research
    • Neuroinflammation and Neurodegeneration Mechanisms
    • Dementia and Cognitive Impairment Research

Papers in

    • Prion Diseases and Protein Misfolding 3
    • S100 Proteins and Annexins 1
    • Amyotrophic Lateral Sclerosis Research 2
    • Traumatic Brain Injury and Neurovascular Disturbances 1

Bart De Vil

7 papers receiving 214 citations

Peers

Bart De Vil
Comparison fields: 5 of 59
  • Neurology 46
  • Neurology 77
  • Psychiatry and Mental health 65
  • Physiology 78
  • Developmental Neuroscience 9
Replace Jonathan M. Schott with:
Jonathan M. Schott United Kingdom
Maria Teresa Dell’Abate Italy
Kaj Blennow Sweden
Antonio Ciacciarelli Italy
Benjamin Saef United States
Emily J. Hill United States
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Citations per field
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Citations per year

Countries citing papers authored by Bart De Vil

Since Specialization
Citations

This map shows the geographic impact of Bart De Vil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bart De Vil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bart De Vil more than expected).

Fields of papers citing papers by Bart De Vil

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bart De Vil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bart De Vil. The network helps show where Bart De Vil may publish in the future.

Co-authors

The 24 scholars most cited alongside Bart De Vil, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Bart De Vil Line = papers co-authored together Bart De Vil links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1 200995
2 201840
3 201634
4 202122
5 201713
6 201811
7 20159

About Bart De Vil

Bart De Vil is a scholar working on Molecular Biology, Neurology, Physiology, Psychiatry and Mental health and Epidemiology, having authored 7 papers that have together received 224 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (4 papers), Dementia and Cognitive Impairment Research (4 papers), Prion Diseases and Protein Misfolding (3 papers), Amyotrophic Lateral Sclerosis Research (2 papers), Schizophrenia research and treatment (1 paper), Acute Ischemic Stroke Management (1 paper), S100 Proteins and Annexins (1 paper) and Traumatic Brain Injury and Neurovascular Disturbances (1 paper). The work is most often cited by research in Neurology (46 citations), Neurology (77 citations), Psychiatry and Mental health (65 citations), Physiology (78 citations) and Developmental Neuroscience (9 citations). Bart De Vil has collaborated with scholars based in Belgium and Netherlands. Frequent co-authors include Patrick Cras, Peter Paul De Deyn, Raf Brouns, Peter Mariën, Didier De Surgeloose, Jean‐Jacques Martin, Sebastiaan Engelborghs, Maria Bjerke, Charisse Somers and Anne Sieben. Their work appears in journals such as Alzheimer s Research & Therapy, Journal of Alzheimer s Disease, Clinical Chemistry, BMC Neurology and Neurobiology of Aging.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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