B.A. Fitscher
Impact in
- Hematology top 5%
- Iron Metabolism and Disorders
- Genetics top 10%
- Hemoglobinopathies and Related Disorders
Papers in
-
- Peroxisome Proliferator-Activated Receptors 5
- Metabolomics and Mass Spectrometry Studies 2
- Oncology 4
- Drug Transport and Resistance Mechanisms 4
- Co-authors
- Wolfgang Stremmel (13 shared papers)H Riedel (7 shared papers)Aline Remus (1 shared paper)Christoph Elsing (2 shared papers)Claudia Denk (2 shared papers)Felix Hoppe‐Seyler (2 shared papers)Irena Crnković‐Mertens (2 shared papers)Karin Butz (2 shared papers)
In The Last Decade
B.A. Fitscher
15 papers receiving 437 citations
Peers
Comparison fields: 5 of 72
- Hematology 148
- Genetics 105
- Nutrition and Dietetics 158
- Clinical Biochemistry 39
- Hepatology 31
Countries citing papers authored by B.A. Fitscher
This map shows the geographic impact of B.A. Fitscher's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by B.A. Fitscher with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites B.A. Fitscher more than expected).
Fields of papers citing papers by B.A. Fitscher
This network shows the impact of papers produced by B.A. Fitscher. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by B.A. Fitscher. The network helps show where B.A. Fitscher may publish in the future.
Co-authors
The 25 scholars most cited alongside B.A. Fitscher, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1999 | 82 | |
| 2 | 1995 | 78 | |
| 3 | 2001 | 67 | |
| 4 | 2000 | 46 | |
| 5 | 1996 | 33 | |
| 6 | 1998 | 33 | |
| 7 | 1997 | 26 | |
| 8 | 1992 | 25 | |
| 9 | 1995 | 17 | |
| 10 | 1990 | 16 | |
| 11 | Fatty acid transporters in plasma membranes of cardiomyocytes in patients with dilated cardiomyopathy. | 2000 | 13 |
| 12 | 2001 | 13 | |
| 13 | 1999 | 8 | |
| 14 | 1991 | 1 | |
| 15 | 2000 | 1 |
About B.A. Fitscher
B.A. Fitscher is a scholar working on Molecular Biology, Oncology, Clinical Biochemistry, Hematology and Genetics, having authored 15 papers that have together received 459 indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (5 papers), Metabolism and Genetic Disorders (4 papers), Drug Transport and Resistance Mechanisms (4 papers), Iron Metabolism and Disorders (3 papers), Hemoglobinopathies and Related Disorders (2 papers), Neonatal Health and Biochemistry (2 papers), Metabolomics and Mass Spectrometry Studies (2 papers) and Trace Elements in Health (2 papers). The work is most often cited by research in Hematology (148 citations), Genetics (105 citations), Nutrition and Dietetics (158 citations), Clinical Biochemistry (39 citations) and Hepatology (31 citations). B.A. Fitscher has collaborated with scholars based in Germany and Italy. Frequent co-authors include Wolfgang Stremmel, H Riedel, Aline Remus, Christoph Elsing, Claudia Denk, Felix Hoppe‐Seyler, Irena Crnković‐Mertens, Karin Butz, Karen Brennan and Sven G. Gehrke. Their work appears in journals such as Blood, Journal of Hepatology, The Journal of Steroid Biochemistry and Molecular Biology, Gastroenterology and Molecular and Cellular Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.