Annick Waldt

8 papers receiving 744 citations

Annick Waldt's Hit Papers

Self-organization and symmetry breaking in intestinal organoid development 2019 · 379 citations
3790+2+4Years since publication100200300

Peers

Annick Waldt
Comparison fields: 5 of 99
  • Cell Biology 125
  • Oncology 191
  • Nephrology 40
  • Immunology 108
  • Hepatology 38
Replace Michael Hannigan with:
Michael Hannigan United States
Marina Lizio Japan
Daniel Dimitrov Germany
Claudia Tulotta Netherlands
Elena Cavallaro Italy
Eitan Wong United States
Angelina J. Lay Australia
Natalia Reglero-Real Spain
Andrés E. Perez Bay United States
Fernando Jose Garcia-Marques United States
Annick Waldt relative to Michael Hannigan United States Michael Hannigan's profile →
Citations per field
00.5×5×10×15×21.8×
Michael Hannigan · 1×
Citations per year

Countries citing papers authored by Annick Waldt

Since Specialization
Citations

This map shows the geographic impact of Annick Waldt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Annick Waldt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Annick Waldt more than expected).

Fields of papers citing papers by Annick Waldt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Annick Waldt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Annick Waldt. The network helps show where Annick Waldt may publish in the future.

Co-authors

The 25 scholars most cited alongside Annick Waldt, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Annick Waldt Line = papers co-authored together Annick Waldt links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1
Self-organization and symmetry breaking in intestinal organoid development
Hit paper breakdown →
2019379
2 2020119
3 202167
4 202159
5 201940
6 202235
7 200726
8 202124
9 20240

About Annick Waldt

Annick Waldt is a scholar working on Molecular Biology, Genetics, Immunology, Nephrology and Pathology and Forensic Medicine, having authored 9 papers that have together received 749 indexed citations. Recurring topics across this work include Renal and related cancers (2 papers), Hippo pathway signaling and YAP/TAZ (1 paper), Advanced Proteomics Techniques and Applications (1 paper), Liver Diseases and Immunity (1 paper), Renal cell carcinoma treatment (1 paper), Cancer Genomics and Diagnostics (1 paper), Chronic Kidney Disease and Diabetes (1 paper) and Inflammation biomarkers and pathways (1 paper). The work is most often cited by research in Cell Biology (125 citations), Oncology (191 citations), Nephrology (40 citations), Immunology (108 citations) and Hepatology (38 citations). Annick Waldt has collaborated with scholars based in Switzerland, United States and China. Frequent co-authors include Guglielmo Roma, Ludivine Challet Meylan, Urs Mayr, Markus Rempfler, Denise Serra, Michael Stadler, Panagiotis Papasaikas, Andrea Boni, Prisca Liberali and Petr Strnad. Their work appears in journals such as Cell stem cell, Neurology Neuroimmunology & Neuroinflammation, Cell Reports, Nature and Sub-cellular biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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