Burkhard Becher

45.9k citations
234 papers · 28.3k · 15 hit papers · h-index 82

Impact in

  • Neurology top 0.01%
    • Neuroinflammation and Neurodegeneration Mechanisms
  • Immunology top 0.02%
    • Immune cells in cancer
    • Immune Cell Function and Interaction
    • T-cell and B-cell Immunology
    • Immunotherapy and Immune Responses
    • Immune Response and Inflammation
    • Psoriasis: Treatment and Pathogenesis

Papers in

    • T-cell and B-cell Immunology 56
    • Immune Cell Function and Interaction 47
    • Immunotherapy and Immune Responses 37
    • Immune Response and Inflammation 34
    • Psoriasis: Treatment and Pathogenesis 32
    • Immune cells in cancer 31
    • Neuroinflammation and Neurodegeneration Mechanisms 57

Burkhard Becher

230 papers receiving 27.9k citations

Burkhard Becher's Hit Papers

Gut-licensed IFNγ+ NK cells drive LAMP1+TRAIL+ anti-inflammatory astrocytes 2021 · 226 citations
2260+7+14Years since publication50010001.5k

Peers

Burkhard Becher
Comparison fields: 5 of 162
  • Neurology 7.0k
  • Immunology 14.9k
  • Biological Psychiatry 1.3k
  • Developmental Neuroscience 1.2k
  • Pathology and Forensic Medicine 3.3k
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Burkhard Becher relative to Ari Waisman Germany Ari Waisman's profile →
Citations per field
00.5×1.5×2.1×
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Citations per year

Countries citing papers authored by Burkhard Becher

Since Specialization
Citations

This map shows the geographic impact of Burkhard Becher's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Burkhard Becher with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Burkhard Becher more than expected).

Fields of papers citing papers by Burkhard Becher

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Burkhard Becher. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Burkhard Becher. The network helps show where Burkhard Becher may publish in the future.

Co-authors

The 25 scholars most cited alongside Burkhard Becher, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Burkhard Becher Line = papers co-authored together Burkhard Becher links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 234 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Immune attack: the role of inflammation in Alzheimer disease
Hit paper breakdown →
20151676
2
Human TH17 lymphocytes promote blood-brain barrier disruption and central nervous system inflammation
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20071428
3
RORγt drives production of the cytokine GM-CSF in helper T cells, which is essential for the effector phase of autoimmune neuroinflammation
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2011966
4
C-Myb+ Erythro-Myeloid Progenitor-Derived Fetal Monocytes Give Rise to Adult Tissue-Resident Macrophages
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2015866
5
High-Dimensional Single-Cell Mapping of Central Nervous System Immune Cells Reveals Distinct Myeloid Subsets in Health, Aging, and Disease
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2018735
6
Dendritic cells permit immune invasion of the CNS in an animal model of multiple sclerosis
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2005725
7
Experimental autoimmune encephalomyelitis repressed by microglial paralysis
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2005623
8
Cytokine networks in neuroinflammation
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2016552
9 2012492
10
IL-1β mediates chronic intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4+ Th17 cells
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2012486
11 2014439
12
Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice
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2012435
13
Single-Cell Mapping of Human Brain Cancer Reveals Tumor-Specific Instruction of Tissue-Invading Leukocytes
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2020434
14
GM-CSF: From Growth Factor to Central Mediator of Tissue Inflammation
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2016432
15
Sall1 is a transcriptional regulator defining microglia identity and function
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2016429
16 2009425
17 2011382
18 2008380
19 2015364
20 2007364

About Burkhard Becher

Burkhard Becher is a scholar working on Immunology, Neurology, Molecular Biology, Oncology and Pathology and Forensic Medicine, having authored 234 papers that have together received 28.3k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (57 papers), T-cell and B-cell Immunology (56 papers), Immune Cell Function and Interaction (47 papers), Immunotherapy and Immune Responses (37 papers), Immune Response and Inflammation (34 papers), Psoriasis: Treatment and Pathogenesis (32 papers), Immune cells in cancer (31 papers) and Multiple Sclerosis Research Studies (23 papers). The work is most often cited by research in Neurology (7.0k citations), Immunology (14.9k citations), Biological Psychiatry (1.3k citations), Developmental Neuroscience (1.2k citations) and Pathology and Forensic Medicine (3.3k citations). Burkhard Becher has collaborated with scholars based in Switzerland, Germany and United States. Frequent co-authors include Frank L. Heppner, Melanie Greter, Richard M. Ransohoff, Jack P. Antel, Randolph J. Noelle, Brigit G. Durell, Alexandre Prat, Sònia Tugues, Andrew L. Croxford and Paulina Kulig. Their work appears in journals such as The Journal of Experimental Medicine, Journal of Clinical Investigation, European Journal of Immunology, Immunity and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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