Annett Eitner

1.4k citations
41 papers · 1.1k · h-index 19

Impact in

    • Osteoarthritis Treatment and Mechanisms
  • Physiology top 10%
    • Pain Mechanisms and Treatments

Papers in

Annett Eitner

38 papers receiving 1.0k citations

Peers

Annett Eitner
Comparison fields: 5 of 98
  • Rheumatology 276
  • Physiology 310
  • Pharmacology 196
  • Behavioral Neuroscience 40
  • Sensory Systems 37
Replace Marcia A. Wheeler with:
Marcia A. Wheeler United States
Manzoor M. Khan United States
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Jun Shimizu Japan
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Citations per field
00.5×7.3×
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Citations per year

Countries citing papers authored by Annett Eitner

Since Specialization
Citations

This map shows the geographic impact of Annett Eitner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Annett Eitner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Annett Eitner more than expected).

Fields of papers citing papers by Annett Eitner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Annett Eitner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Annett Eitner. The network helps show where Annett Eitner may publish in the future.

Co-authors

The 25 scholars most cited alongside Annett Eitner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Annett Eitner Line = papers co-authored together Annett Eitner links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 41 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2017170
2 2007126
3 201077
4 201365
5 201763
6 201348
7 200939
8 201638
9 201535
10 202034
11 201429
12 201127
13 200727
14 200825
15 202125
16 200721
17 202119
18 201619
19 202218
20 201516

About Annett Eitner

Annett Eitner is a scholar working on Physiology, Cellular and Molecular Neuroscience, Rheumatology, Molecular Biology and Pharmacology, having authored 41 papers that have together received 1.1k indexed citations. Recurring topics across this work include Pain Mechanisms and Treatments (17 papers), Osteoarthritis Treatment and Mechanisms (9 papers), Neuropeptides and Animal Physiology (7 papers), Inflammatory mediators and NSAID effects (6 papers), Stress Responses and Cortisol (5 papers), Pharmacological Effects of Natural Compounds (5 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers) and Neuroscience and Neuropharmacology Research (4 papers). The work is most often cited by research in Rheumatology (276 citations), Physiology (310 citations), Pharmacology (196 citations), Behavioral Neuroscience (40 citations) and Sensory Systems (37 citations). Annett Eitner has collaborated with scholars based in Germany, United States and Australia. Frequent co-authors include Hans‐Georg Schaible, Gunther O. Hofmann, Reimar Krieg, Roland Zell, Andi Krumbholz, Peter Wutzler, Karl-Jürgen Halbhuber, Andrea Ebersberger, Gisela Segond von Banchet and Frank Richter. Their work appears in journals such as Osteoarthritis and Cartilage, Arthritis & Rheumatology, International Journal of Molecular Sciences, Pain and Acta Histochemica.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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