Anna Leichsenring

409 citations
15 papers · 355 · h-index 9

Impact in

  • Physiology top 2%
    • Adenosine and Purinergic Signaling
    • Pain Mechanisms and Treatments
  • Neurology top 10%
    • Neuroinflammation and Neurodegeneration Mechanisms

Papers in

Anna Leichsenring

14 papers receiving 348 citations

Peers

Anna Leichsenring
Comparison fields: 5 of 68
  • Physiology 119
  • Neurology 71
  • Biological Psychiatry 14
  • Developmental Neuroscience 23
  • Endocrine and Autonomic Systems 36
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Marı́a Antonia Baltrons Spain
Shao Xia Lin United States
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Michelle C. Catlin United States
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Citations per field
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Citations per year

Countries citing papers authored by Anna Leichsenring

Since Specialization
Citations

This map shows the geographic impact of Anna Leichsenring's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anna Leichsenring with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anna Leichsenring more than expected).

Fields of papers citing papers by Anna Leichsenring

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anna Leichsenring. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anna Leichsenring. The network helps show where Anna Leichsenring may publish in the future.

Co-authors

The 25 scholars most cited alongside Anna Leichsenring, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Anna Leichsenring Line = papers co-authored together Anna Leichsenring links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 201075
2 201668
3 201157
4 200935
5 201631
6 200623
7 201216
8 200815
9 201110
10 20128
11 20147
12 20177
13 20162
14 20201
15 20240

About Anna Leichsenring

Anna Leichsenring is a scholar working on Physiology, Endocrine and Autonomic Systems, Neurology, Cellular and Molecular Neuroscience and Immunology, having authored 15 papers that have together received 355 indexed citations. Recurring topics across this work include Adenosine and Purinergic Signaling (5 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (3 papers), Immune Response and Inflammation (3 papers), Neuroscience of respiration and sleep (3 papers), Knowledge Management and Sharing (2 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers), Pharmacological Receptor Mechanisms and Effects (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). The work is most often cited by research in Physiology (119 citations), Neurology (71 citations), Biological Psychiatry (14 citations), Developmental Neuroscience (23 citations) and Endocrine and Autonomic Systems (36 citations). Anna Leichsenring has collaborated with scholars based in Germany, Hungary and Czechia. Frequent co-authors include Péter Illés, Patrizia Rubini, Thomas Riedel, László Köles, Christine C. Stichel, Hermann Lübbert, Franziska Lange, Wolfgang Nörenberg, Claudia Heine and Ute Krügel. Their work appears in journals such as PLoS ONE, Innate Immunity, Journal of Immunological Methods, Naunyn-Schmiedeberg s Archives of Pharmacology and Advances in pharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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