Amol Ugale

470 citations
8 papers · 355 · h-index 6

Impact in

  • Aging top 5%
    • Genetics, Aging, and Longevity in Model Organisms
  • Hematology top 5%
    • Hematopoietic Stem Cell Transplantation
    • Acute Myeloid Leukemia Research

Papers in

    • Epigenetics and DNA Methylation 3
    • DNA Repair Mechanisms 1
    • Ubiquitin and proteasome pathways 1
    • Protein Degradation and Inhibitors 1
    • Acute Myeloid Leukemia Research 4
    • Hematopoietic Stem Cell Transplantation 3

Amol Ugale

8 papers receiving 352 citations

Peers

Amol Ugale
Comparison fields: 5 of 47
  • Aging 41
  • Hematology 167
  • Immunology 70
  • Molecular Biology 231
  • Genetics 27
Replace Andrew DeVilbiss with:
Andrew DeVilbiss United States
Tiancheng Fang United States
Seka Lazare Netherlands
Nicholas Stong United States
Ellen L. van Agtmaal Netherlands
Damià Romero–Moya Spain
K Ohyashiki Japan
A.J. Montandon United Kingdom
Imme Krüger Germany
Óscar Navarro-Montero Spain
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Citations per field
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Citations per year

Countries citing papers authored by Amol Ugale

Since Specialization
Citations

This map shows the geographic impact of Amol Ugale's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amol Ugale with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amol Ugale more than expected).

Fields of papers citing papers by Amol Ugale

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amol Ugale. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amol Ugale. The network helps show where Amol Ugale may publish in the future.

Co-authors

The 24 scholars most cited alongside Amol Ugale, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Amol Ugale Line = papers co-authored together Amol Ugale links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 2011201
2 201378
3 201441
4 201513
5 201712
6 20166
7 20222
8 20242

About Amol Ugale

Amol Ugale is a scholar working on Molecular Biology, Hematology, Immunology, Public Health, Environmental and Occupational Health and Infectious Diseases, having authored 8 papers that have together received 355 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (4 papers), Epigenetics and DNA Methylation (3 papers), Hematopoietic Stem Cell Transplantation (3 papers), Immune Cell Function and Interaction (2 papers), DNA Repair Mechanisms (1 paper), Acute Lymphoblastic Leukemia research (1 paper), Ubiquitin and proteasome pathways (1 paper) and Protein Degradation and Inhibitors (1 paper). The work is most often cited by research in Aging (41 citations), Hematology (167 citations), Immunology (70 citations), Molecular Biology (231 citations) and Genetics (27 citations). Amol Ugale has collaborated with scholars based in Sweden, Austria and Spain. Frequent co-authors include David Bryder, Martin Wahlestedt, Gudmundur L. Norddahl, Mikael Sigvardsson, Cornelis Jan Pronk, Jens Nygren, Tomas Deierborg, Ragnar Mattsson, Petter Säwén and Shamit Soneji. Their work appears in journals such as The Journal of Cell Biology, Cell Reports, Cell stem cell, Leukemia and Blood.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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