Alice Emptoz
Impact in
- Sensory Systems top 0.5%
- Hearing, Cochlea, Tinnitus, Genetics
- Neurology top 10%
- Vestibular and auditory disorders
Papers in
-
- Hearing, Cochlea, Tinnitus, Genetics 7
-
- RNA and protein synthesis mechanisms 1
- Ion channel regulation and function 1
- Co-authors
- Saaïd Safieddine (7 shared papers)Christine Petit (7 shared papers)Didier Dulon (6 shared papers)Jacques Boutet de Monvel (3 shared papers)Jean‐Pierre Hardelin (3 shared papers)Omar Akil (3 shared papers)Sylvie Nouaille (3 shared papers)Lawrence R. Lustig (3 shared papers)
- Journals
- Proceedings of the National Academy of Sciences (2 papers)Journal of Neuroscience (2 papers)Cell (1 paper)Journal of Clinical Investigation (1 paper)eLife (1 paper)
- Partner nations
- FranceUnited StatesHungary
In The Last Decade
Alice Emptoz
7 papers receiving 723 citations
Peers
Comparison fields: 5 of 57
- Sensory Systems 516
- Neurology 100
- Otorhinolaryngology 44
- Cognitive Neuroscience 140
- Molecular Biology 423
Countries citing papers authored by Alice Emptoz
This map shows the geographic impact of Alice Emptoz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alice Emptoz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alice Emptoz more than expected).
Fields of papers citing papers by Alice Emptoz
This network shows the impact of papers produced by Alice Emptoz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alice Emptoz. The network helps show where Alice Emptoz may publish in the future.
Co-authors
The 25 scholars most cited alongside Alice Emptoz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 210 | |
| 2 | 2015 | 159 | |
| 3 | 2017 | 108 | |
| 4 | 2018 | 103 | |
| 5 | 2017 | 87 | |
| 6 | 2019 | 34 | |
| 7 | 2017 | 30 |
About Alice Emptoz
Alice Emptoz is a scholar working on Sensory Systems, Molecular Biology, Nutrition and Dietetics, Ecology and Cognitive Neuroscience, having authored 7 papers that have together received 731 indexed citations. Recurring topics across this work include Hearing, Cochlea, Tinnitus, Genetics (7 papers), Biochemical Analysis and Sensing Techniques (2 papers), Hearing Loss and Rehabilitation (1 paper), Marine animal studies overview (1 paper), RNA and protein synthesis mechanisms (1 paper), Cancer-related molecular mechanisms research (1 paper), Ion channel regulation and function (1 paper) and Vestibular and auditory disorders (1 paper). The work is most often cited by research in Sensory Systems (516 citations), Neurology (100 citations), Otorhinolaryngology (44 citations), Cognitive Neuroscience (140 citations) and Molecular Biology (423 citations). Alice Emptoz has collaborated with scholars based in France, United States and Hungary. Frequent co-authors include Saaïd Safieddine, Christine Petit, Didier Dulon, Jacques Boutet de Monvel, Jean‐Pierre Hardelin, Omar Akil, Sylvie Nouaille, Lawrence R. Lustig, Ghizlène Lahlou and William W. Hauswirth. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience, Cell, Journal of Clinical Investigation and eLife.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.