Aiko Itoh
Impact in
- Immunology top 5%
- Galectins and Cancer Biology
- Toxin Mechanisms and Immunotoxins
- Immune Cell Function and Interaction
- Macrophage Migration Inhibitory Factor
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- Signaling Pathways in Disease
- Glycosylation and Glycoproteins Research
- Protein Tyrosine Phosphatases
Papers in
- Immunology 12
- Galectins and Cancer Biology 12
- Toxin Mechanisms and Immunotoxins 4
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- Signaling Pathways in Disease 6
- Glycosylation and Glycoproteins Research 6
- Protein Tyrosine Phosphatases 1
- Co-authors
- Takanori Nakamura (11 shared papers)Nozomu Nishi (11 shared papers)Hiroki Shoji (4 shared papers)Mitsuomi Hirashima (3 shared papers)Hiroshi Miyanaka (3 shared papers)Masako Seki (1 shared paper)Naoko Yoshida (1 shared paper)Shinichi Araya (1 shared paper)
In The Last Decade
Aiko Itoh
13 papers receiving 638 citations
Peers
Comparison fields: 5 of 57
- Immunology 607
- Molecular Biology 400
- Oncology 105
- Immunology and Allergy 11
- Pathology and Forensic Medicine 20
Countries citing papers authored by Aiko Itoh
This map shows the geographic impact of Aiko Itoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aiko Itoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aiko Itoh more than expected).
Fields of papers citing papers by Aiko Itoh
This network shows the impact of papers produced by Aiko Itoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aiko Itoh. The network helps show where Aiko Itoh may publish in the future.
Co-authors
The 25 scholars most cited alongside Aiko Itoh, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2005 | 173 | |
| 2 | 2003 | 140 | |
| 3 | 2005 | 129 | |
| 4 | 2008 | 61 | |
| 5 | 2006 | 54 | |
| 6 | 2012 | 38 | |
| 7 | 2018 | 14 | |
| 8 | 2013 | 12 | |
| 9 | 2020 | 8 | |
| 10 | 2014 | 6 | |
| 11 | 2015 | 4 | |
| 12 | 2017 | 4 | |
| 13 | 1998 | 2 |
About Aiko Itoh
Aiko Itoh is a scholar working on Immunology, Molecular Biology, Oncology, Cellular and Molecular Neuroscience and Biomaterials, having authored 13 papers that have together received 645 indexed citations. Recurring topics across this work include Galectins and Cancer Biology (12 papers), Signaling Pathways in Disease (6 papers), Glycosylation and Glycoproteins Research (6 papers), Peptidase Inhibition and Analysis (4 papers), Toxin Mechanisms and Immunotoxins (4 papers), Magnesium Alloys: Properties and Applications (1 paper), Hereditary Neurological Disorders (1 paper) and Protein Tyrosine Phosphatases (1 paper). The work is most often cited by research in Immunology (607 citations), Molecular Biology (400 citations), Oncology (105 citations), Immunology and Allergy (11 citations) and Pathology and Forensic Medicine (20 citations). Aiko Itoh has collaborated with scholars based in Japan, China and Russia. Frequent co-authors include Takanori Nakamura, Nozomu Nishi, Hiroki Shoji, Mitsuomi Hirashima, Hiroshi Miyanaka, Masako Seki, Naoko Yoshida, Shinichi Araya, Hiromi Yoshida and S. Kamitori. Their work appears in journals such as Glycobiology, The Journal of Immunology, FEBS Letters, Journal of Biological Chemistry and FEBS Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.