A.C. Kato

402 citations
18 papers · 370 · h-index 11

Impact in

Papers in

A.C. Kato

18 papers receiving 336 citations

Peers

A.C. Kato
Comparison fields: 5 of 63
  • Developmental Neuroscience 60
  • Cellular and Molecular Neuroscience 233
  • Neurology 51
  • Neurology 28
  • Pharmacology 52
Replace D. L. McIlwain with:
D. L. McIlwain United States
Carin A. Loewen United States
J. Schonbach Switzerland
H. El‐Bizri Canada
Aldo Calliari Uruguay
Amy Aylsworth Canada
Erika Lorenzetto Italy
Miguel A. Chinchetru Spain
Anna Aurora Battaglia United Kingdom
Carina Weissmann Argentina
A.C. Kato relative to D. L. McIlwain United States D. L. McIlwain's profile →
Citations per field
00.5×2×3.3×
D. L. McIlwain · 1×
Citations per year

Countries citing papers authored by A.C. Kato

Since Specialization
Citations

This map shows the geographic impact of A.C. Kato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A.C. Kato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A.C. Kato more than expected).

Fields of papers citing papers by A.C. Kato

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A.C. Kato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A.C. Kato. The network helps show where A.C. Kato may publish in the future.

Co-authors

The 18 scholars most cited alongside A.C. Kato, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with A.C. Kato Line = papers co-authored together A.C. Kato links everyone, so they are left out of the graph.

All Works

18 of 18 papers shown
#Work
1 198377
2 198957
3 198040
4 198327
5 197726
6 198226
7 198326
8 198219
9 199017
10 197712
11 199010
12 198910
13 199110
14 19934
15 19923
16 19903
17 19872
18 19841

About A.C. Kato

A.C. Kato is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Developmental Neuroscience, Pharmacology and Neurology, having authored 18 papers that have together received 370 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (9 papers), Nerve injury and regeneration (6 papers), Neurogenesis and neuroplasticity mechanisms (5 papers), Neuroscience and Neural Engineering (4 papers), Cholinesterase and Neurodegenerative Diseases (4 papers), Amyotrophic Lateral Sclerosis Research (3 papers), Ion channel regulation and function (2 papers) and Pluripotent Stem Cells Research (2 papers). The work is most often cited by research in Developmental Neuroscience (60 citations), Cellular and Molecular Neuroscience (233 citations), Neurology (51 citations), Neurology (28 citations) and Pharmacology (52 citations). A.C. Kato has collaborated with scholars based in Switzerland and France. Frequent co-authors include Daniel Bertrand, C R Bader, Élisabeth Dupin, Linda Erkman, L. Wuarin, M. Rey, Yves Dunant, Bernard W. Fulpius, Jean‐Claude Martinou and M.I. Glavinoviċ. Their work appears in journals such as Developmental Biology, Developmental Brain Research, Stereotactic and Functional Neurosurgery, Biochemical and Biophysical Research Communications and Brain Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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