A.‐C. EKLÖF

18 papers receiving 459 citations

Peers

A.‐C. EKLÖF
Comparison fields: 5 of 61
  • Nephrology 56
  • Endocrinology, Diabetes and Metabolism 128
  • Clinical Biochemistry 31
  • Cardiology and Cardiovascular Medicine 88
  • Biochemistry 28
Replace R. Kirsten with:
R. Kirsten Germany
Xinqun Hu United States
M. Mello-Aires Brazil
Michelle Iacovitti Canada
Catherine Vernimmen France
Y. L. Chan United States
Motoei Kunimi Japan
Sten Christensen Denmark
Jessica Fryckstedt Sweden
V. Marinescu United States
A.‐C. EKLÖF relative to R. Kirsten Germany R. Kirsten's profile →
Citations per field
00.5×2.7×
R. Kirsten · 1×
Citations per year

Countries citing papers authored by A.‐C. EKLÖF

Since Specialization
Citations

This map shows the geographic impact of A.‐C. EKLÖF's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A.‐C. EKLÖF with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A.‐C. EKLÖF more than expected).

Fields of papers citing papers by A.‐C. EKLÖF

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A.‐C. EKLÖF. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A.‐C. EKLÖF. The network helps show where A.‐C. EKLÖF may publish in the future.

Co-authors

The 25 scholars most cited alongside A.‐C. EKLÖF, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with A.‐C. EKLÖF Line = papers co-authored together A.‐C. EKLÖF links everyone, so they are left out of the graph.

All Works

19 of 19 papers shown
#Work
1 1993113
2 199679
3 199364
4 200062
5 199729
6 198715
7 199215
8 199715
9 199213
10 198612
11 200112
12 19918
13 19867
14 19896
15 20066
16 19925
17 19905
18 20103
19 20090

About A.‐C. EKLÖF

A.‐C. EKLÖF is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine, Pediatrics, Perinatology and Child Health, Pulmonary and Respiratory Medicine and Endocrinology, Diabetes and Metabolism, having authored 19 papers that have together received 469 indexed citations. Recurring topics across this work include Ion Transport and Channel Regulation (6 papers), Renin-Angiotensin System Studies (5 papers), Hormonal Regulation and Hypertension (5 papers), Birth, Development, and Health (4 papers), Renal and Vascular Pathologies (3 papers), Aldose Reductase and Taurine (3 papers), Neuroendocrine regulation and behavior (2 papers) and Dialysis and Renal Disease Management (2 papers). The work is most often cited by research in Nephrology (56 citations), Endocrinology, Diabetes and Metabolism (128 citations), Clinical Biochemistry (31 citations), Cardiology and Cardiovascular Medicine (88 citations) and Biochemistry (28 citations). A.‐C. EKLÖF has collaborated with scholars based in Sweden, United States and Hungary. Frequent co-authors include Anita Aperia, Akinori Nishi, Alejandro M. Bertorello, Paul Greengard, Gianni Celsi, Fernando R. Ibarra, Anna Körner, Leif Svensson, Roger Belusa and Georg Jaremko. Their work appears in journals such as Diabetologia, Hypertension, Diabetes, Proceedings of the National Academy of Sciences and Clinical and Experimental Hypertension.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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