A. Ottolini

21 papers receiving 301 citations

Peers

A. Ottolini
Comparison fields: 5 of 59
  • Psychiatry and Mental health 84
  • Cellular and Molecular Neuroscience 71
  • Endocrine and Autonomic Systems 20
  • Genetics 27
  • Neurology 15
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Countries citing papers authored by A. Ottolini

Since Specialization
Citations

This map shows the geographic impact of A. Ottolini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Ottolini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Ottolini more than expected).

Fields of papers citing papers by A. Ottolini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Ottolini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Ottolini. The network helps show where A. Ottolini may publish in the future.

Co-authors

The 25 scholars most cited alongside A. Ottolini, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with A. Ottolini Line = papers co-authored together A. Ottolini links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 23 papers — load more, or switch the sort, to bring in the rest.

#Work
1 199448
2 198344
3 201340
4 198830
5
[Congenital muscular dystrophy with merosin deficiency: clinical, histopathological, immunocytochemical and genetic analysis].
199630
6 199127
7 198923
8 201522
9 199110
10 20169
11 19939
12 20087
13
The efficacy of noninvasive mechanical ventilation on nocturnal hypoxaemia in Duchenne's muscular dystrophy.
19987
14 19976
15
Myotonic dystrophy in childhood.
19825
16
Computerized cranial tomography in Wilson disease: report of a case before and after penicillamine therapy.
19813
17
Classification of neuromuscular diseases.
19932
18 19901
19 19901
20 20091

About A. Ottolini

A. Ottolini is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Genetics, Pediatrics, Perinatology and Child Health and Endocrine and Autonomic Systems, having authored 23 papers that have together received 327 indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (3 papers), Neurogenetic and Muscular Disorders Research (3 papers), Muscle Physiology and Disorders (3 papers), Neuroscience of respiration and sleep (3 papers), DNA Repair Mechanisms (2 papers), Myasthenia Gravis and Thymoma (2 papers), Adipose Tissue and Metabolism (2 papers) and Autism Spectrum Disorder Research (2 papers). The work is most often cited by research in Psychiatry and Mental health (84 citations), Cellular and Molecular Neuroscience (71 citations), Endocrine and Autonomic Systems (20 citations), Genetics (27 citations) and Neurology (15 citations). A. Ottolini has collaborated with scholars based in Italy, United Kingdom and Ireland. Frequent co-authors include G. Lanzi, Raffaele Manni, Umberto Balottin, Pierangelo Veggiotti, Elisa Fazzi, Francesco Pasquali, Emanuela Maserati, Giovanni Lanzi, Isa Cerveri and A Martelli. Their work appears in journals such as Journal of Computer Assisted Tomography, Journal of Attention Disorders, Child s Nervous System, Neural Plasticity and Cephalalgia.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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