A. Goepfert

771 citations
16 papers · 604 · h-index 10

Impact in

Papers in

    • Psoriasis: Treatment and Pathogenesis 3
    • Toxin Mechanisms and Immunotoxins 2
    • Bartonella species infections research 2

A. Goepfert

14 papers receiving 588 citations

Peers

A. Goepfert
Comparison fields: 5 of 73
  • Endocrinology 108
  • Molecular Medicine 45
  • Parasitology 51
  • Immunology 155
  • Oncology 109
Replace Kinlin L. Chao with:
Kinlin L. Chao United States
Birte Hernandez Alvarez Germany
Eva Bjur Sweden
Nathan P. Manes United States
Isabel Rodríguez‐Escudero Spain
Shuangping Shi United States
Isabelle Ricard France
Elias R. Gerrick United States
Danielle Ensergueix France
Maxime Wéry France
A. Goepfert relative to Kinlin L. Chao United States Kinlin L. Chao's profile →
Citations per field
00.5×2×2.6×
Kinlin L. Chao · 1×
Citations per year

Countries citing papers authored by A. Goepfert

Since Specialization
Citations

This map shows the geographic impact of A. Goepfert's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Goepfert with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Goepfert more than expected).

Fields of papers citing papers by A. Goepfert

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Goepfert. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Goepfert. The network helps show where A. Goepfert may publish in the future.

Co-authors

The 25 scholars most cited alongside A. Goepfert, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with A. Goepfert Line = papers co-authored together A. Goepfert links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 2012134
2 201585
3 201776
4 202063
5 200962
6 201148
7 201540
8 201331
9 202224
10 199611
11 19999
12 20218
13 20127
14 19976
15 20250
16 20160

About A. Goepfert

A. Goepfert is a scholar working on Immunology, Parasitology, Oncology, Molecular Biology and Pathology and Forensic Medicine, having authored 16 papers that have together received 604 indexed citations. Recurring topics across this work include Psoriasis: Treatment and Pathogenesis (3 papers), Protein Hydrolysis and Bioactive Peptides (3 papers), Toxin Mechanisms and Immunotoxins (2 papers), Signaling Pathways in Disease (2 papers), Epigenetics and DNA Methylation (2 papers), Whipple's Disease and Interleukins (2 papers), Bartonella species infections research (2 papers) and Enzyme Production and Characterization (1 paper). The work is most often cited by research in Endocrinology (108 citations), Molecular Medicine (45 citations), Parasitology (51 citations), Immunology (155 citations) and Oncology (109 citations). A. Goepfert has collaborated with scholars based in Switzerland, Germany and United Kingdom. Frequent co-authors include Christoph Dehio, Tilman Schirmer, Frédéric V. Stanger, Sylvie Lehmann, Jean‐Michel Rondeau, Alexander Harms, Philipp Engel, Alexander Schmidt, Frank Kolbinger and Jutta Blank. Their work appears in journals such as Cell Reports, Nature, Annals of Oncology, Scientific Reports and mAbs.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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