Ze‐Yi Lim

1.3k citations
8 papers · 854 · h-index 8

Impact in

Papers in

    • Protein Kinase Regulation and GTPase Signaling 4
    • Signaling Pathways in Disease 1
    • Cellular transport and secretion 5
    • Hippo pathway signaling and YAP/TAZ 1

Ze‐Yi Lim

8 papers receiving 847 citations

Peers

Ze‐Yi Lim
Comparison fields: 5 of 73
  • Cell Biology 354
  • Physiology 60
  • Biochemistry 79
  • Molecular Biology 563
  • Immunology 160
Replace Cristiano Iurisci with:
Cristiano Iurisci Italy
Wendy K. Greentree United States
Carmen De Lemos-Chiarandini United States
R F Weber United States
Zoi Erpapazoglou France
Hisao Nagaya Japan
Yan Shan Ong Singapore
Javier Valdez Taubas Argentina
Jonathan D. Gary United States
Nicolas Touchot France
Ze‐Yi Lim relative to Cristiano Iurisci Italy Cristiano Iurisci's profile →
Citations per field
00.5×1.5×2.1×
Cristiano Iurisci · 1×
Citations per year

Countries citing papers authored by Ze‐Yi Lim

Since Specialization
Citations

This map shows the geographic impact of Ze‐Yi Lim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ze‐Yi Lim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ze‐Yi Lim more than expected).

Fields of papers citing papers by Ze‐Yi Lim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ze‐Yi Lim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ze‐Yi Lim. The network helps show where Ze‐Yi Lim may publish in the future.

Co-authors

The 25 scholars most cited alongside Ze‐Yi Lim, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ze‐Yi Lim Line = papers co-authored together Ze‐Yi Lim links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 2001342
2 2004169
3 2001111
4 200191
5 200952
6 200036
7 200235
8 200118

About Ze‐Yi Lim

Ze‐Yi Lim is a scholar working on Molecular Biology, Cell Biology, Surgery, Oncology and Physiology, having authored 8 papers that have together received 854 indexed citations. Recurring topics across this work include Cellular transport and secretion (5 papers), Protein Kinase Regulation and GTPase Signaling (4 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (1 paper), Pancreatic function and diabetes (1 paper), Signaling Pathways in Disease (1 paper), Hippo pathway signaling and YAP/TAZ (1 paper), Calcium signaling and nucleotide metabolism (1 paper) and Blood disorders and treatments (1 paper). The work is most often cited by research in Cell Biology (354 citations), Physiology (60 citations), Biochemistry (79 citations), Molecular Biology (563 citations) and Immunology (160 citations). Ze‐Yi Lim has collaborated with scholars based in United Kingdom, United States and Australia. Frequent co-authors include Andrew B. Holmes, Nicholas T. Ktistakis, Jan Willem Thuring, Phillip T. Hawkins, Len Stephens, Maria Manifava, Karen E. Anderson, John Coadwell, Paul Tempst and Hediye Erdjument‐Bromage. Their work appears in journals such as Nature Cell Biology, Chemical Communications, Current Biology, Journal of Biological Chemistry and Journal of Cell Science.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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