Wei Qü
Impact in
- Cancer Research top 10%
- Cancer-related molecular mechanisms research
- MicroRNA in disease regulation
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- Epigenetics and DNA Methylation
- RNA modifications and cancer
- Genomics and Phylogenetic Studies
Papers in
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- Epigenetics and DNA Methylation 9
- RNA modifications and cancer 7
- Cancer-related gene regulation 4
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- Nerve injury and regeneration 6
- Co-authors
- Shinichi Morishita (13 shared papers)Shinichi Hashimoto (6 shared papers)Sumio Sugano (7 shared papers)Kouji Matsushima (5 shared papers)Yoichiro Nakatani (5 shared papers)Yiheng He (2 shared papers)Jingping Rao (2 shared papers)Shoukun Han (2 shared papers)
- Journals
- Scientific Reports (3 papers)BMC Genomics (2 papers)Genome Research (2 papers)BioMed Research International (1 paper)PLoS ONE (1 paper)
- Partner nations
- ChinaJapanUnited States
In The Last Decade
Wei Qü
72 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 112
- Cancer Research 152
- Molecular Biology 439
- Immunology 84
- Reproductive Medicine 31
- Plant Science 147
Countries citing papers authored by Wei Qü
This map shows the geographic impact of Wei Qü's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Qü with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Qü more than expected).
Fields of papers citing papers by Wei Qü
This network shows the impact of papers produced by Wei Qü. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Qü. The network helps show where Wei Qü may publish in the future.
Co-authors
The 25 scholars most cited alongside Wei Qü, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 76 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 57 | |
| 2 | Interleukin 7 signaling prevents apoptosis by regulating bcl-2 and bax via the p53 pathway in human non-small cell lung cancer cells. | 2014 | 51 |
| 3 | 2012 | 48 | |
| 4 | 2016 | 47 | |
| 5 | 2009 | 47 | |
| 6 | 2018 | 45 | |
| 7 | 2011 | 43 | |
| 8 | 2013 | 39 | |
| 9 | 2016 | 38 | |
| 10 | 2008 | 37 | |
| 11 | 2020 | 35 | |
| 12 | 2012 | 35 | |
| 13 | 2014 | 34 | |
| 14 | 2020 | 30 | |
| 15 | 2020 | 30 | |
| 16 | 2023 | 21 | |
| 17 | 2016 | 21 | |
| 18 | Emodin inhibits HMGB1-induced tumor angiogenesis in human osteosarcoma by regulating SIRT1. | 2015 | 21 |
| 19 | 2020 | 21 | |
| 20 | 2020 | 20 |
About Wei Qü
Wei Qü is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Pulmonary and Respiratory Medicine, Genetics and Plant Science, having authored 76 papers that have together received 1.0k indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (9 papers), RNA modifications and cancer (7 papers), Nerve injury and regeneration (6 papers), Neurogenesis and neuroplasticity mechanisms (4 papers), Cancer-related gene regulation (4 papers), Neurofibromatosis and Schwannoma Cases (3 papers), Breast Cancer Treatment Studies (3 papers) and HER2/EGFR in Cancer Research (3 papers). The work is most often cited by research in Cancer Research (152 citations), Molecular Biology (439 citations), Immunology (84 citations), Reproductive Medicine (31 citations) and Plant Science (147 citations). Wei Qü has collaborated with scholars based in China, Japan and United States. Frequent co-authors include Shinichi Morishita, Shinichi Hashimoto, Sumio Sugano, Kouji Matsushima, Yoichiro Nakatani, Yiheng He, Jingping Rao, Shoukun Han, Jing Zhang and Yufei Wang. Their work appears in journals such as Scientific Reports, BMC Genomics, Genome Research, BioMed Research International and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.