Wayne Chadwick

1.3k citations
31 papers · 1.1k · h-index 19

Impact in

Papers in

    • Receptor Mechanisms and Signaling 6
    • Mitochondrial Function and Pathology 4
    • Adipose Tissue and Metabolism 6
    • Alzheimer's disease research and treatments 4

Wayne Chadwick

31 papers receiving 1.1k citations

Peers

Wayne Chadwick
Comparison fields: 5 of 106
  • Endocrine and Autonomic Systems 128
  • Cellular and Molecular Neuroscience 314
  • Biological Psychiatry 34
  • Aging 22
  • Sensory Systems 52
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Citations per field
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Citations per year

Countries citing papers authored by Wayne Chadwick

Since Specialization
Citations

This map shows the geographic impact of Wayne Chadwick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wayne Chadwick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wayne Chadwick more than expected).

Fields of papers citing papers by Wayne Chadwick

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wayne Chadwick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wayne Chadwick. The network helps show where Wayne Chadwick may publish in the future.

Co-authors

The 25 scholars most cited alongside Wayne Chadwick, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Wayne Chadwick Line = papers co-authored together Wayne Chadwick links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 31 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2008156
2 201092
3 201187
4 201068
5 201264
6 200863
7 200663
8 201057
9 201050
10 201236
11 200935
12 201135
13 201230
14 201228
15 201026
16 201626
17 201126
18 201420
19 201120
20 201018

About Wayne Chadwick

Wayne Chadwick is a scholar working on Molecular Biology, Physiology, Cellular and Molecular Neuroscience, Endocrine and Autonomic Systems and Spectroscopy, having authored 31 papers that have together received 1.1k indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (6 papers), Adipose Tissue and Metabolism (6 papers), Alzheimer's disease research and treatments (4 papers), Advanced Proteomics Techniques and Applications (4 papers), Circadian rhythm and melatonin (4 papers), Mitochondrial Function and Pathology (4 papers), Neuropeptides and Animal Physiology (4 papers) and Genetic Neurodegenerative Diseases (3 papers). The work is most often cited by research in Endocrine and Autonomic Systems (128 citations), Cellular and Molecular Neuroscience (314 citations), Biological Psychiatry (34 citations), Aging (22 citations) and Sensory Systems (52 citations). Wayne Chadwick has collaborated with scholars based in United States, Belgium and United Kingdom. Frequent co-authors include Stuart Maudsley, Bronwen Martin, Sung Soo Park, Liyun Wang, Kevin G. Becker, Mark P. Mattson, Randall Brenneman, Yu Zhou, Josephine M. Egan and Olga D. Carlson. Their work appears in journals such as PLoS ONE, Diabetes, CNS & Neurological Disorders - Drug Targets, Journal of Biological Chemistry and BMC Neuroscience.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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