W. Witt

21 papers receiving 363 citations

Peers

W. Witt
Comparison fields: 5 of 68
  • Internal Medicine 43
  • Hematology 74
  • Cancer Research 90
  • Cardiology and Cardiovascular Medicine 103
  • Pharmacology 72
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Countries citing papers authored by W. Witt

Since Specialization
Citations

This map shows the geographic impact of W. Witt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. Witt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. Witt more than expected).

Fields of papers citing papers by W. Witt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. Witt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. Witt. The network helps show where W. Witt may publish in the future.

Co-authors

The 25 scholars most cited alongside W. Witt, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with W. Witt Line = papers co-authored together W. Witt links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 22 papers — load more, or switch the sort, to bring in the rest.

#Work
1 198663
2 199448
3 199246
4 198740
5 198235
6 199234
7 199233
8
Adherence of leucocytes to electrically damaged venules in vivo. Effects of iloprost, PGE1, indomethacin, forskolin, BW 755 C, sulotroban, hirudin, and thrombocytopenia.
198823
9 199317
10
Antithrombotic profile of iloprost in experimental models of in vivo platelet aggregation and thrombosis.
198716
11 199513
12 198811
13 198810
14
Potential therapeutic mechanisms of stable prostacyclin (PGI2)-mimetics in severe peripheral vascular disease.
198810
15
Effects of prostacyclin analogues in in vivo tumor models.
199110
16 19947
17 19884
18
Lexikon der Kartographie
19793
19 19863
20 19873

About W. Witt

W. Witt is a scholar working on Pulmonary and Respiratory Medicine, Pharmacology, Cardiology and Cardiovascular Medicine, Internal Medicine and Analytical Chemistry, having authored 22 papers that have together received 430 indexed citations. Recurring topics across this work include Blood properties and coagulation (7 papers), Analytical Methods in Pharmaceuticals (6 papers), Venous Thromboembolism Diagnosis and Management (6 papers), Antiplatelet Therapy and Cardiovascular Diseases (5 papers), Cancer Treatment and Pharmacology (3 papers), Protease and Inhibitor Mechanisms (3 papers), Venomous Animal Envenomation and Studies (3 papers) and Blood Coagulation and Thrombosis Mechanisms (3 papers). The work is most often cited by research in Internal Medicine (43 citations), Hematology (74 citations), Cancer Research (90 citations), Cardiology and Cardiovascular Medicine (103 citations) and Pharmacology (72 citations). W. Witt has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include Peter Donner, S Stürzebecher, Peter Bringmann, Linda Cashion, Wolf‐Dieter Schleuning, E. Schillinger, Werner Skuballa, Michael S. Hildebrand, Bettina Müller and Martin Haberey. Their work appears in journals such as Prostaglandins, Blood, Blood Coagulation & Fibrinolysis, Radiology and Annals of the New York Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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